Pacientes submetidos à adrenalectomia unilateral para aldosteronismo primário (AP) unilateral
Este procedimento leva à cura da hipertensão em 50% a 60% dos pacientes cuidadosamente selecionados e melhora em todos os demais.[27]Stowasser M, Gordon RD. Primary aldosteronism - careful investigation is essential and rewarding. Mol Cell Endocrinol. 2004 Mar 31;217(1-2):33-9.
http://www.ncbi.nlm.nih.gov/pubmed/15134798?tool=bestpractice.com
[106]Stowasser M, Gordon RD, Gunasekera TG, et al. High rate of detection of primary aldosteronism, including surgically treatable forms, after 'non-selective' screening of hypertensive patients. J Hypertens. 2003 Nov;21(11):2149-57.
http://www.ncbi.nlm.nih.gov/pubmed/14597859?tool=bestpractice.com
[115]Celen O, O'Brien MJ, Melby JC, et al. Factors influencing outcome of surgery for primary aldosteronism. Arch Surg. 1996 Jun;131(6):646-50.
http://www.ncbi.nlm.nih.gov/pubmed/8645073?tool=bestpractice.com
[116]Rutherford JC, Taylor WL, Stowasser M, et al. Success of surgery in primary aldosteronism judged by residual autonomous aldosterone production. World J Surg. 1998 Dec;22(12):1243-5.
http://www.ncbi.nlm.nih.gov/pubmed/9841751?tool=bestpractice.com
[117]Stowasser M, Klemm SA, Tunny TJ, et al. Response to unilateral adrenalectomy for aldosterone-producing adenoma - effect of potassium levels and angiotensin responsiveness. Clin Exp Pharmacol Physiol. 1994 Apr;21(4):319-22.
http://www.ncbi.nlm.nih.gov/pubmed/7923899?tool=bestpractice.com
Normalmente, a PA se normaliza ou mostra melhora máxima em 1 a 6 meses após a adrenalectomia unilateral, mas pode continuar caindo por até 1 a 2 anos em alguns pacientes.[6]Stowasser M, Gordon RD, Rutherford JC, et al. Diagnosis and management of primary aldosteronism. J Renin Angiotensin Aldosterone Syst. 2001 Sep;2(3):156-69.
http://journals.sagepub.com/doi/pdf/10.3317/jraas.2001.022
http://www.ncbi.nlm.nih.gov/pubmed/11881117?tool=bestpractice.com
O aldosteronismo primário (AP) é curado bioquimicamente em 70% dos pacientes totalmente investigados e melhora em todos os demais pacientes.[27]Stowasser M, Gordon RD. Primary aldosteronism - careful investigation is essential and rewarding. Mol Cell Endocrinol. 2004 Mar 31;217(1-2):33-9.
http://www.ncbi.nlm.nih.gov/pubmed/15134798?tool=bestpractice.com
[106]Stowasser M, Gordon RD, Gunasekera TG, et al. High rate of detection of primary aldosteronism, including surgically treatable forms, after 'non-selective' screening of hypertensive patients. J Hypertens. 2003 Nov;21(11):2149-57.
http://www.ncbi.nlm.nih.gov/pubmed/14597859?tool=bestpractice.com
[116]Rutherford JC, Taylor WL, Stowasser M, et al. Success of surgery in primary aldosteronism judged by residual autonomous aldosterone production. World J Surg. 1998 Dec;22(12):1243-5.
http://www.ncbi.nlm.nih.gov/pubmed/9841751?tool=bestpractice.com
Menos de 20% dos pacientes necessitam de doses equivalentes ou aumentadas de medicamentos após a cirurgia.[103]Yip L, Duh QY, Wachtel H, et al. American Association of Endocrine Surgeons guidelines for adrenalectomy: executive summary. JAMA Surg. 2022 Oct 1;157(10):870-7.
https://jamanetwork.com/journals/jamasurgery/fullarticle/2795363
http://www.ncbi.nlm.nih.gov/pubmed/35976622?tool=bestpractice.com
Há uma melhora consistente na qualidade de vida. Estudos de coorte têm mostrado melhoras significativas nos parâmetros cardiovasculares (incluindo massa ventricular esquerda na ecocardiografia).[61]Rossi GP, Sacchetto A, Visentin P, et al. Changes in left ventricular anatomy and function in hypertension and primary aldosteronism. Hypertension. 1996 May;27(5):1039-45.
http://hyper.ahajournals.org/content/27/5/1039.full
http://www.ncbi.nlm.nih.gov/pubmed/8621194?tool=bestpractice.com
[125]Catena C, Colussi GL, Lapenna R, et al. Long-term cardiac effects of adrenalectomy or mineralocorticoid antagonists in patients with primary aldosteronism. Hypertension. 2007 Nov;50(5):911-8.
http://hyper.ahajournals.org/content/50/5/911.full
http://www.ncbi.nlm.nih.gov/pubmed/17893375?tool=bestpractice.com
A recorrência de AP nas pessoas aparentemente curadas após 12 meses é incomum. Nos pacientes com AP persistente (embora melhorado), a hipertensão pode responder bem a pequenas doses de medicamentos antagonistas da aldosterona, mas deve-se ter cuidado porque é provável que os níveis de aldosterona tenham sido significativamente reduzidos.[6]Stowasser M, Gordon RD, Rutherford JC, et al. Diagnosis and management of primary aldosteronism. J Renin Angiotensin Aldosterone Syst. 2001 Sep;2(3):156-69.
http://journals.sagepub.com/doi/pdf/10.3317/jraas.2001.022
http://www.ncbi.nlm.nih.gov/pubmed/11881117?tool=bestpractice.com
Pacientes submetidos ao tratamento com medicamentos antagonistas de aldosterona
A hipertensão melhora e é controlada na maioria dos casos.[27]Stowasser M, Gordon RD. Primary aldosteronism - careful investigation is essential and rewarding. Mol Cell Endocrinol. 2004 Mar 31;217(1-2):33-9.
http://www.ncbi.nlm.nih.gov/pubmed/15134798?tool=bestpractice.com
[106]Stowasser M, Gordon RD, Gunasekera TG, et al. High rate of detection of primary aldosteronism, including surgically treatable forms, after 'non-selective' screening of hypertensive patients. J Hypertens. 2003 Nov;21(11):2149-57.
http://www.ncbi.nlm.nih.gov/pubmed/14597859?tool=bestpractice.com
[122]Lim PO, Young WF, MacDonald TM. A review of the medical treatment of primary aldosteronism. J Hypertens. 2001 Mar;19(3):353-61.
http://www.ncbi.nlm.nih.gov/pubmed/11288803?tool=bestpractice.com
A hipocalemia, quando presente, quase sempre é corrigida.
No entanto, as melhoras não são tão marcantes quanto depois da adrenalectomia unilateral para lesões de lateralização. Em estudos de coorte, o número médio de medicamentos anti-hipertensivos necessários não caiu muito, e houve uma melhora menos surpreendente na massa ventricular esquerda derivada por ecocardiografia.[27]Stowasser M, Gordon RD. Primary aldosteronism - careful investigation is essential and rewarding. Mol Cell Endocrinol. 2004 Mar 31;217(1-2):33-9.
http://www.ncbi.nlm.nih.gov/pubmed/15134798?tool=bestpractice.com
[61]Rossi GP, Sacchetto A, Visentin P, et al. Changes in left ventricular anatomy and function in hypertension and primary aldosteronism. Hypertension. 1996 May;27(5):1039-45.
http://hyper.ahajournals.org/content/27/5/1039.full
http://www.ncbi.nlm.nih.gov/pubmed/8621194?tool=bestpractice.com
Pacientes com hiperaldosteronismo familiar tipo I (HF-I) submetidos ao tratamento com medicamentos glicocorticoides
A hipertensão no hiperaldosteronismo familiar tipo I (HF-I) costuma ser de início precoce e grave o suficiente para causar morte precoce, geralmente por acidente vascular cerebral (AVC) hemorrágico, a não ser que seja especificamente tratada.[20]Stowasser M, Gartside MG, Gordon RD. A PCR-based method of screening individuals of all ages, from neonates to the elderly, for familial hyperaldosteronism type I. Aust N Z J Med. 1997 Dec;27(6):685-90.
http://www.ncbi.nlm.nih.gov/pubmed/9483237?tool=bestpractice.com
[21]Rich GM, Ulick S, Cook S, et al. Glucocorticoid-remediable aldosteronism in a large kindred: clinical spectrum and diagnosis using a characteristic biochemical phenotype. Ann Intern Med. 1992 May 15;116(10):813-20.
http://www.ncbi.nlm.nih.gov/pubmed/1567095?tool=bestpractice.com
No entanto, o tratamento com glicocorticoides, administrados em doses baixas que não causem efeitos adversos cushingoides, geralmente é bastante efetivo no controle da hipertensão (evitando assim o AVC), sendo anti-hipertensivos suplementares necessários apenas ocasionalmente.[132]Walker BR, Edwards CR. Dexamethasone-suppressible hypertension. Endocrinologist. 1993 Mar;3(2):87-97.
http://journals.lww.com/theendocrinologist/Abstract/1993/03000/Dexamethasone_Suppressible_Hypertension_.3.aspx