A prevalência da síndrome de hipoventilação varia com a causa subjacente. Há relatos de que aproximadamente 10% a 20% dos pacientes com apneia obstrutiva do sono (AOS) apresentam síndrome de hipoventilação na obesidade (SHO).[2]Mokhlesi B, Tulaimat A, Faibussowitsch I, et al. Obesity hypoventilation syndrome: prevalence and predictors in patients with obstructive sleep apnea. Sleep Breath. 2007;11:117-124.
http://www.ncbi.nlm.nih.gov/pubmed/17187265?tool=bestpractice.com
[3]Kessler R, Chaouat A, Schinkewitch P, et al. The obesity-hypoventilation syndrome revisited: a prospective study of 34 consecutive cases. Chest. 2001;120:369-376.
http://journal.publications.chestnet.org/article.aspx?articleid=1079891
http://www.ncbi.nlm.nih.gov/pubmed/11502631?tool=bestpractice.com
[4]Lecube A, Sampol G, Lloberes P, et al. Asymptomatic sleep-disordered breathing in premenopausal women awaiting bariatric surgery. Obes Surg. 2010;20:454-461.
http://www.ncbi.nlm.nih.gov/pubmed/20020222?tool=bestpractice.com
Porém, aproximadamente 10% dos pacientes com SHO apresentam um índice de apneia-hipopneia normal, com <5 eventos/hora.[2]Mokhlesi B, Tulaimat A, Faibussowitsch I, et al. Obesity hypoventilation syndrome: prevalence and predictors in patients with obstructive sleep apnea. Sleep Breath. 2007;11:117-124.
http://www.ncbi.nlm.nih.gov/pubmed/17187265?tool=bestpractice.com
[3]Kessler R, Chaouat A, Schinkewitch P, et al. The obesity-hypoventilation syndrome revisited: a prospective study of 34 consecutive cases. Chest. 2001;120:369-376.
http://journal.publications.chestnet.org/article.aspx?articleid=1079891
http://www.ncbi.nlm.nih.gov/pubmed/11502631?tool=bestpractice.com
A respiração de Cheyne-Stokes é relatada em 33% a 42% dos pacientes que têm insuficiência cardíaca congestiva e fração de ejeção do ventrículo esquerdo <45%, com uma prevalência de até 56% nos pacientes hospitalizados que estão aguardando transplante cardíaco.[5]Javaheri S, Parker TJ, Liming JD, et al. Sleep apnea in 81 ambulatory male patients with stable heart failure: types and their prevalences, consequences, and presentations. Circulation. 1998;97:2154-2159.
http://circ.ahajournals.org/cgi/content/full/97/21/2154
http://www.ncbi.nlm.nih.gov/pubmed/9626176?tool=bestpractice.com
[6]Sin DD, Fitzgerald F, Parker JD, et al. Risk factors for central and obstructive sleep apnea in 450 men and women with congestive heart failure. Am J Respir Crit Care Med. 1999;160:1101-1106.
http://www.atsjournals.org/doi/full/10.1164/ajrccm.160.4.9903020
http://www.ncbi.nlm.nih.gov/pubmed/10508793?tool=bestpractice.com
[7]Oldenburg O, Lamp B, Faber L, et al. Sleep-disordered breathing in patients with symptomatic heart failure: a contemporary study of prevalence in and characteristics of 700 patients. Eur J Heart Fail. 2007;9:251-257.
http://www.ncbi.nlm.nih.gov/pubmed/17027333?tool=bestpractice.com
[8]Krachman SL, D'Alonzo GE, Berger TJ, et al. Comparison of oxygen therapy with nasal continuous positive airway pressure on Cheyne-Stokes respiration during sleep in congestive heart failure. Chest. 1999;116:1550-1557.
http://journal.publications.chestnet.org/article.aspx?articleid=1078359
http://www.ncbi.nlm.nih.gov/pubmed/10593775?tool=bestpractice.com
Cerca de 27% dos pacientes com doença pulmonar obstrutiva crônica (DPOC) subjacente desenvolveram hipoxemia noturna, mas somente aqueles com AOS associada (chamada de síndrome de sobreposição) ou com obstrução grave (volume expiratório forçado no primeiro segundo [VEF1] <35% do predito) podem apresentar hipercapnia associada. Nos pacientes com distúrbios torácicos restritivos, como as deformidades da parede torácica (por exemplo, cifoescoliose), a prevalência de hipoventilação depende do grau de curvatura espinhal (ângulo de Cobb >120°). Já os pacientes com doenças neuromusculares podem, em algum momento, desenvolver a síndrome de hipoventilação, dependendo da doença subjacente (por exemplo, distrofia muscular de Duchenne).