History and exam
Key diagnostic factors
common
chest pain
dyspnea
The degree of dyspnea depends on the hypoxia caused and presence and severity of pre-existing lung disease. It does not necessarily correlate to the size of the pneumothorax.[1]
hyperexpanded ipsilateral hemithorax
The degree of hyperexpansion depends on the size of the pneumothorax and whether a tension pneumothorax develops.[1]
hyper-resonant ipsilateral hemithorax
Hyper-resonance on percussion of the affected side.
ipsilateral absent or diminished breath sounds
Diminished or absent breath sounds on the affected side.
uncommon
Risk factors
strong
cigarette smoking
family history of pneumothorax
tall and slender body build
age <40 years
The peak age for primary spontaneous pneumothorax is 20 years at the first episode. Primary spontaneous pneumothoraces rarely occur after 40 years of age.[13]
recent invasive medical procedure
Invasive procedures such as transcutaneous needle aspiration of lung lesions, thoracentesis, bronchoscopic transbronchial biopsy, central venous catheter placement, and positive pressure ventilation are associated with iatrogenic pneumothoraces.
acute severe asthma
The air trapping associated with airway inflammation during an asthmatic attack can cause rupture of alveolar sacs leading to the development of a pneumothorax.[1]
COPD
This is the leading cause of secondary spontaneous pneumothoraces and is due to rupture of subpleural emphysematous blebs.[18]
tuberculosis
Secondary spontaneous pneumothoraces occur in 1.5% of cases of active pulmonary tuberculosis. Ruptures of subpleural tuberculous cysts are thought to be responsible.[21]
AIDS-related Pneumocystis jirovecii infection
cystic fibrosis
Secondary spontaneous pneumothorax is a frequent occurrence in cystic fibrosis and is associated with more severe disease. About 16% to 20% of patients with cystic fibrosis >18 years of age will experience a pneumothorax at some time in their lives. Recurrent contralateral pneumothoraces occur in 40% of patients.[28][29]
lymphangioleiomyomatosis
A multisystem disease of women, characterized by cystic lung destruction that can result in recurrent pneumothoraces.[30]
Birt-Hogg-Dube syndrome
An autosomal dominant inheritable disease characterized by pulmonary cysts, spontaneous pneumothoraces, benign skin lesions, and renal cancers. Mutations in the gene that encodes for folliculin have been identified in individuals with this familial spontaneous pneumothorax.[31]
pulmonary Langerhans cell histiocytosis
This is a smoking-related interstitial lung disease, characterized by the development of cystic changes in the lung that predisposes to pneumothorax.[32]
Erdheim-Chester disease
A rare disease characterized by disseminated non-Langerhans cell histiocytosis involving multiple organs. Pulmonary involvement is uncommon but the lung can become infiltrated by lipid-laden histiocytes, resulting in diffuse interstitial cystic changes and pneumothorax.[33]
weak
Marfan syndrome
There are reports of families afflicted with Marfan syndrome whose members suffered multiple bilateral episodes of primary spontaneous pneumothoraces. In this population, primary spontaneous pneumothoraces are attributed to pulmonary tissue fragility related to defective fibrillin.[14]
homocystinuria
There have been a few case reports of primary spontaneous pneumothoraces in patients with homocystinuria. The pathophysiology of this association is unknown.[17]
primary lung cancer and metastatic cancer to the lungs
Pneumothorax can occur in bronchogenic carcinomas and in a variety of cancers that have metastasized to the lungs. The pneumothoraces can develop following chemotherapy. It is postulated that necrosis of the peripherally located cancer causes the tumor to rupture into the pleural space, resulting in a pneumothorax.[19][20]
Use of this content is subject to our disclaimer