Differentials

Drug rash with eosinophilia and systemic symptoms (DRESS)

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Presents with fever, rash, lymphadenopathy, arthralgias, hepatitis, nephritis, carditis, eosinophilia, and atypical lymphocytes.[19][66][67]

Most frequent causative agents are phenytoin, phenobarbital and carbamazepine, and the sulfonamides.[19][68]

More frequent among black people.

It takes 2-6 weeks from beginning the drug therapy to onset of rash.[3]

INVESTIGATIONS

Skin biopsy and clinical presentation; CBC may show eosinophilia; LFTs may be elevated if hepatitis present.

Staphylococcal scalded skin syndrome

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Disease of infancy or early childhood (Ritter disease).

Bacterial endotoxin disrupts the epidermis but without necrosis or inflammation.[24]

INVESTIGATIONS

CBC, blood and wound cultures, and skin biopsy.

Toxic shock syndrome

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Either Staphylococcus or Streptococcus can be the origin of the infection, presenting with signs of sepsis and a sunburn-like rash.

INVESTIGATIONS

CBC, blood and wound cultures, and skin biopsy.

Graft-versus-host disease

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Patients develop a rash, but the skin in graft-versus-host disease does not slough off as readily as it does in SJS or TEN.

Patients who have been transplanted develop a rash as a result of rejection rather than a drug-induced reaction, although grade IV skin manifestations can be as severe as in TEN. Ocular lesions are rare.[24]

INVESTIGATIONS

CBC and skin biopsy.

Vesicant/blister agent poisoning

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Clinical effects such as erythema and blisters will occur immediately with phosgene oxime or lewisite or be delayed for 2-24 hours with mustard gas.[69]

INVESTIGATIONS

Skin biopsy.

Erythema multiforme

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Target lesions normally triggered by herpes viruses.[3][20]

INVESTIGATIONS

Skin biopsy.

Burns, skin

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Result of trauma with destruction of the epidermis and dermis to variable degrees.[19]

INVESTIGATIONS

Skin biopsy if there is not a good history.

Paraneoplastic pemphigus

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Blistering disease with a malignant comorbidity.[19]

INVESTIGATIONS

Skin biopsy and direct immunofluorescence.

Generalized bullous fixed drug eruption

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

History of recurrence in same area when rechallenged with offending drug; few, if any, systemic symptoms; round erythematous or blister patches.[70][71]

INVESTIGATIONS

Skin biopsy.

Acute generalized exanthematous pustulosis

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Fever, neutrophilia, acute sterile pustules, erythema, facial edema.[72]

INVESTIGATIONS

Skin biopsy.

Coxsackievirus A6

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

No drug intake; fever; papulovesicles on hands, tongue, buttocks, mouth ulcers.[73]

INVESTIGATIONS

Skin biopsy; virus in skin and blister fluid.

Chikungunya fever

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Resolves usually within 1 week. Vesicles; bullae; few smaller, superficial erosions. Mucositis generally absent; if present, mild erosions that heal in 3 or 4 days. Child active, playful, eating well. Nelson severity index score 8-10 (moderately sick or not sick).[74]

INVESTIGATIONS

IgM anti-chikungunya virus antibodies; histopathology; Nelson severity index.

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