Investigations

1st investigations to order

basic metabolic profile (including urea and creatinine)

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Result
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Order as soon as possible and as routine follow-up in at-risk patients.

Creatinine is not a reliable indicator of renal failure as sudden decrease in glomerular filtration rate can over-estimate creatinine.

Changes in creatinine and urea levels can be influenced by other non-renal events such as rhabdomyolysis, nutritional state, use of steroids, or bleeding.[5]

Result

elevated serum creatinine, elevated urea, hyperkalaemia, or metabolic acidosis suggests ATN

urea-to-creatinine ratio

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Result
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In cases of ATN the ratio of blood urea to creatinine falls to 10:1, as tubular injury means there is no increased reabsorption of water, sodium, and urea.[2][5]

Result

10:1 supports ATN (in a pre-renal situation, the ratio can rise to 20:1 or higher)[2]

urine sodium concentration

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Result
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Tubule dysfunction leads to increased urinary sodium concentration.[5]

Result

elevated (>40 mmol/L [40 mEq/L])

urine osmolality

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Result
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Impairment in urinary concentrating capacity is characterised by decrease in urine osmolality.[5]

Can help differentiate between pre-renal azotaemia (in which the re-absorptive capacity and concentrating ability of the kidney are preserved or enhanced) and ATN (in which these functions are impaired).

Result

<450 mOsmol/kg supports ATN

fractional excretion of sodium

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Result
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Tubule dysfunction leads to increased fractional excretion of sodium.[5]

Low sensitivity and specificity for ATN. Also present in haemolysis, sepsis, cirrhosis, and chronic heart failure.

Result

over 2% supports ATN

fractional excretion of urea

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Result
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More reliable in patients taking diuretics or with chronic kidney disease.

Result

over 35% supports ATN[29]

urinalysis for sediment

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Result
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Can help distinguish from pre-renal azotaemia.[39]

Haem-positive urine in the absence of erythrocytes in the sediment suggests ATN from haemolysis or rhabdomyolysis.

Result

tubular epithelial cells, epithelial cell casts, or muddy brown casts supports ATN

FBC

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Result
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Mild to moderate anaemia is commonly observed in some types of ATN, such as in multiple myeloma, bleeding, haemolysis, or chronic kidney disease.

Result

anaemia

coagulation studies

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Result
Test

Prolonged ATN can result in bleeding as a result of dysfunctional platelets.

Result

prolonged PTT

urinary myoglobin

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Result
Test

Elevated myoglobin levels suggest ATN from rhabdomyolysis.

Result

elevated

Investigations to consider

arterial blood gases (ABG)

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Result
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Assists in further evaluation of acidosis, which is often suggested by the low bicarbonate on the basic metabolic profile.

Result

metabolic acidosis

platelet aggregation studies

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Result
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Prolonged ATN can result in bleeding as a result of platelet dysfunction.

Result

prolonged

ultrasound

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Result
Test

Assists in evaluation of post-obstructive causes as well as in the evaluation of renal architecture and size (underlying chronic kidney disease).

Result

normal in ATN

ECG

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Result
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May occur with hyperkalaemia.

Performed if hyperkalaemia is suspected or detected by laboratory tests.

Result

arrhythmias, peaked T waves, increased PR interval, widened QRS, atrial arrest, and deterioration to a sine wave pattern (if severe hyperkalaemia)

inferior vena cava (IVC) diameter measured by ultrasound

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Result
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Perform to assess intravascular volume status.

Result

IVC diameter >2.5 cm suggests volume overload; IVC diameter <1.5 cm suggests volume depletion[30]

renal biopsy

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Result
Test

Should only be performed when the history, clinical features, and findings of laboratory and radiological investigation suggest primary renal disease other than ischaemic or toxic-related ATN.[5]

Result

patchy and focal loss of individual cells or clusters of cells from tubular epithelium with resultant gaps, and exposure of areas of denudated basement membrane

serum and urinary biomarkers

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Result
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Various novel serum and urinary biomarkers are showing potential as useful indicators for the diagnosis and classification of acute kidney injury.[31][32] Kidney injury molecule-1 (KIM-1) has been shown to be a good early biomarker of ischaemic ATN.[33][34] Other biomarkers include neutrophil gelatinase-associated lipocalin (NGAL), interleukin-18 (IL-18), tissue inhibitor of metalloproteinase-2 (TIMP-2), and insulin-like growth factor binding protein 7 (IGFBP7).[35][36][37]

Result

results indicative of renal damage; confined to research studies, not widely applied to clinical settings[38]

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