Differentials

Food poisoning

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Predominant symptom is vomiting.

It is easy to confuse the symptoms of TD with those of food poisoning; the latter is of much earlier onset and is characterised more by vomiting than by diarrhoea (except that caused by Clostridium perfringens).

Food poisoning, while self-limited, does not respond to antibiotics.

Staphylococcus aureus and Bacillus cereus (both forming heat-stable toxins) or C perfringens (heat-labile toxins) cause most food poisoning cases.

Pre-formed toxins (from S aureus or B cereus) produce symptoms (vomiting > diarrhoea) within 1-6 hours, whereas C perfringens infections, with in vivo toxin formation, cause diarrhoeal symptoms within 8-16 hours. Most TD bacterial infections, on the other hand, become symptomatic 16 hours after ingestion.[20]

INVESTIGATIONS

Typically diagnosed clinically, but bacterial cultures or virological studies may elicit culprit organism.

Irritable bowel syndrome

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Diarrhoea or constipation, both associated with abdominal pain. No weight loss, fever, or systemic symptoms. Symptom relief usually occurs after bowel movement. Post-TD irritable bowel syndrome (IBS) is usually of the diarrhoea subtype (IBS-D).[6]

INVESTIGATIONS

Normal examination, and laboratory and bowel work-up.

Secondary disaccharidase (or other dietary) deficiency

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Exacerbation of symptoms with dairy products or other food class. Often difficult to solicit. Generally resolves in days to a week.

INVESTIGATIONS

Normal examination and laboratory and bowel work-up

Malabsorptive conditions

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Persistent diarrhoea, may be unusually foul smelling or greasy. Can be attributed to small intestinal bacterial overgrowth, or changes in microbiome or in structural absorptive capacity of microvilli.

INVESTIGATIONS

Faecal fat, faecal bile acid, D-xylose, hydrogen/methane breath testing, colonoscopy/endoscopy may be revealing.

Clostridioides difficile colitis

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Persisting diarrhoea with fever, abdominal pain/tenderness, and weight loss, often following use of antibiotics (with or without travel history).

INVESTIGATIONS

C difficile stool toxin positive (enzyme immunoassay/polymerase chain reaction tests are available).

Coeliac disease

SIGNS / SYMPTOMS
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SIGNS / SYMPTOMS

Persisting diarrhoea with malabsorption (with or without travel history).

May be associated with dermatitis herpetiformis.

INVESTIGATIONS

IgA anti-tissue transglutaminase (anti-tTG) usually positive.

Because IgA deficiency may interfere with coeliac testing, an IgA level should also be obtained.

Crohn's disease

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Diarrhoea (with or without travel history), abdominal pain, fever, perianal fistulae.

INVESTIGATIONS

Elevated erythrocyte sedimentation rate, elevated faecal calprotectin, anaemia, haem-positive stools. Colonoscopy differentiates most cases of Crohn's disease from ulcerative colitis. Ulcerative colitis always involves the rectum and is contiguous versus intermittent. Crohn's disease often has perianal involvement, rectal sparing, and a tendency to form fistulae.

Ulcerative colitis

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Bloody diarrhoea (with or without travel history), abdominal pain, fever, no perianal disease.

INVESTIGATIONS

Elevated erythrocyte sedimentation rate, elevated faecal calprotectin, anaemia, haem-positive stools. Colonoscopy differentiates most cases of Crohn's disease from ulcerative colitis. Ulcerative colitis always involves the rectum and is contiguous versus intermittent. Terminal ileitis may be present in ulcerative colitis with pancolitis due to backwash.

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