Patients should be evaluated annually to judge adequacy of glucocorticoid and mineralocorticoid replacement.[2]Bornstein SR, Allolio B, Arlt W, et al. Diagnosis and treatment of primary adrenal insufficiency: an Endocrine Society clinical practice guideline. J Clin Endocrinol Metab. 2016 Feb;101(2):364-89.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4880116
http://www.ncbi.nlm.nih.gov/pubmed/26760044?tool=bestpractice.com
Annual laboratory examination should include serum electrolytes measurement to monitor mineralocorticoid replacement.
Glucocorticoid adjustments are made according to signs and symptoms. Continuance of excessive fatigue and increasing pigmentation after start of treatment are suggestive of insufficient glucocorticoid replacement. Conversely, weight gain or facial plethora suggests glucocorticoid excess.
Mineralocorticoid replacement should be tapered down if the patient presents with fluid retention, hypertension, decreased renin activity, or hypokalaemia, and increased if there is postural hypotension or hyperkalaemia.
Long-term over-replacement of glucocorticoid may be associated with decreased bone density. Regular measurements of bone mineral density are suggested.[3]Husebye ES, Pearce SH, Krone NP, et al. Adrenal insufficiency. Lancet. 2021 Feb 13;397(10274):613-29.
http://www.ncbi.nlm.nih.gov/pubmed/33484633?tool=bestpractice.com
In patients with autoimmune adrenalitis, surveillance for other associated autoimmune diseases is suggested.[2]Bornstein SR, Allolio B, Arlt W, et al. Diagnosis and treatment of primary adrenal insufficiency: an Endocrine Society clinical practice guideline. J Clin Endocrinol Metab. 2016 Feb;101(2):364-89.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4880116
http://www.ncbi.nlm.nih.gov/pubmed/26760044?tool=bestpractice.com
[4]Hahner S, Ross RJ, Arlt W, et al. Adrenal insufficiency. Nat Rev Dis Primers. 2021 Mar 11;7(1):19.
http://www.ncbi.nlm.nih.gov/pubmed/33707469?tool=bestpractice.com
[35]Betterle C, Dal Pra C, Mantero F, et al. Autoimmune adrenal insufficiency and autoimmune polyendocrine syndromes: autoantibodies, autoantigens, and their applicability in diagnosis and disease prediction. Endocr Rev. 2002 Jun;23(3):327-64.
https://academic.oup.com/edrv/article/23/3/327/2424246
http://www.ncbi.nlm.nih.gov/pubmed/12050123?tool=bestpractice.com
Commonly associated conditions include autoimmune thyroid disease, autoimmune gastritis, type 1 diabetes, premature ovarian failure, vitiligo, or coeliac disease.[9]Betterle C, Presotto F, Furmaniak J. Epidemiology, pathogenesis, and diagnosis of Addison's disease in adults. J Endocrinol Invest. 2019 Dec;42(12):1407-33.
http://www.ncbi.nlm.nih.gov/pubmed/31321757?tool=bestpractice.com