Other presentations
PPD is most commonly seen in people with schizophrenia, but may present in those with other psychiatric disorders and neurodevelopmental disorders.[1]Verghese C, De Leon J, Josiassen RC. Problems and progress in the diagnosis and treatment of polydipsia and hyponatremia. Schizophr Bull. 1996;22(3):455-64.
https://academic.oup.com/schizophreniabulletin/article/22/3/455/1829836
http://www.ncbi.nlm.nih.gov/pubmed/8873296?tool=bestpractice.com
[2]de Leon J. Polydipsia: a study in a long-term psychiatric unit. Eur Arch Psychiatry Clin Neurosci. 2003 Feb;253(1):37-9.
http://www.ncbi.nlm.nih.gov/pubmed/12664312?tool=bestpractice.com
[3]Illowsky BP, Kirch DG. Polydipsia and hyponatremia in psychiatric patients. Am J Psychiatry. 1988 Jun;145(6):675-83.
http://www.ncbi.nlm.nih.gov/pubmed/3285701?tool=bestpractice.com
[4]Jose CJ, Perez-Cruet J. Incidence and morbidity of self-induced water intoxication in state mental hospital patients. Am J Psychiatry. 1979 Feb;136(2):221-2.
http://www.ncbi.nlm.nih.gov/pubmed/760555?tool=bestpractice.com
[5]Blum A, Tempey FW, Lynch WJ. Somatic findings in patients with psychogenic polydipsia. J Clin Psychiatry. 1983 Feb;44(2):55-6.
http://www.ncbi.nlm.nih.gov/pubmed/6826531?tool=bestpractice.com
[6]Meulendijks D, Mannesse CK, Jansen PA, et al. Antipsychotic-induced hyponatraemia: a systematic review of the published evidence. Drug Saf. 2010 Feb 1;33(2):101-14.
http://www.ncbi.nlm.nih.gov/pubmed/20082537?tool=bestpractice.com
[7]Mannesse CK, van Puijenbroek EP, Jansen PA, et al. Hyponatraemia as an adverse drug reaction of antipsychotic drugs: a case-control study in VigiBase. Drug Saf. 2010 Jul 1;33(7):569-78.
http://www.ncbi.nlm.nih.gov/pubmed/20553058?tool=bestpractice.com
Polydipsia may be diagnosed by an incidental finding of low plasma sodium (hyponatremia), or by overt symptoms of water intoxication necessitating medical intervention.[8]Hariprasad MK, Eisinger RP, Nadler IM, et al. Hyponatremia in psychogenic polydipsia. Arch Intern Med. 1980 Dec;140(12):1639-42.
http://www.ncbi.nlm.nih.gov/pubmed/7458496?tool=bestpractice.com
[9]Jose CJ, Barton JL, Perez-Cruet J. Hyponatremic seizures in psychiatric patients. Biol Psychiatry. 1979 Oct;14(5):839-43.
http://www.ncbi.nlm.nih.gov/pubmed/497307?tool=bestpractice.com
Polydipsia can occur in the absence of underlying medical or psychiatric conditions.[10]Ahmadi L, Goldman MB. Primary polydipsia: update. Best Pract Res Clin Endocrinol Metab. 2020 Sep;34(5):101469.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7683824
http://www.ncbi.nlm.nih.gov/pubmed/33222764?tool=bestpractice.com
Increasing popularity of lifestyle programs that advocate water drinking to improve overall health have led to a rise in polydipsia among the general population.[10]Ahmadi L, Goldman MB. Primary polydipsia: update. Best Pract Res Clin Endocrinol Metab. 2020 Sep;34(5):101469.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7683824
http://www.ncbi.nlm.nih.gov/pubmed/33222764?tool=bestpractice.com
Chronic excessive hydration may not manifest neurologically, due to volume adaptation by the brain.[11]Janicic N, Verbalis JG. Evaluation and management of hypo-osmolality in hospitalized patients. Endocrinol Metab Clin North Am. 2003 Jun;32(2):459-81, vii.
http://www.ncbi.nlm.nih.gov/pubmed/12800541?tool=bestpractice.com
Between 3% and 6% of patients with PPD and schizophrenia have hyponatremia.[10]Ahmadi L, Goldman MB. Primary polydipsia: update. Best Pract Res Clin Endocrinol Metab. 2020 Sep;34(5):101469.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7683824
http://www.ncbi.nlm.nih.gov/pubmed/33222764?tool=bestpractice.com
[12]Gleadhill IC, Smith TA, Yium JJ. Hyponatremia in patients with schizophrenia. South Med J. 1982 Apr;75(4):426-8.
http://www.ncbi.nlm.nih.gov/pubmed/6122272?tool=bestpractice.com
With mildly reduced serum sodium levels, symptoms may range from nausea and malaise to lethargy, headache, obtundation, seizures, and coma. Large and rapid fluid intake can lead to severe and symptomatic hyponatremia. At very low serum sodium levels (<115 mEq/L), neurologic symptoms manifest, due to intracerebral osmotic fluid shifts and cerebral edema. In the most severe cases, progression to tentorial herniation, brain stem compression, and respiratory arrest can result in death.