Differentials

Flat warts

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Flesh-colored, flat-topped, slightly raised papules.

Usually multiple and found on the extremities or face.

INVESTIGATIONS

No differentiating tests.

Palmoplantar warts

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Palmoplantar warts are endophytic and occasionally tender, with scale and black dots on the surface, and are commonly found on the palms and soles.

INVESTIGATIONS

No differentiating tests.

Seborrheic keratosis

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Lesions are grayish-brown, stuck-on, scaly papules with white pseudocysts and a smooth or rough surface.

Many are present in the same skin area and persist indefinitely.

INVESTIGATIONS

No differentiating tests.

Acquired digital fibrokeratoma

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Lesions occur on the digit, and have a firm, smooth surface.

There are no tiny black dots on the surface.

INVESTIGATIONS

No differentiating tests.

Clavus

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Lesions occur on the digit in the areas of friction and over bony prominences and have a firm, rough surface.

There are no tiny black dots on the surface.

INVESTIGATIONS

Skin biopsy shows hyperkeratosis and parakeratosis with central epidermal atrophy, plus peripheral acanthosis and possible perivascular infiltrates of the upper dermis.

Callus

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Lesions occur on the digit in the areas of friction and over bony prominences and have a firm, rough surface.

There are no tiny black dots on the surface.

INVESTIGATIONS

Skin biopsy shows hyperkeratosis.

Squamous cell carcinoma

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Nonhealing, enlarging, hyperkeratotic papule that becomes inflamed and tender.

INVESTIGATIONS

Skin biopsy shows parakeratosis, atypical keratinocytes with or without koilocytes.

There may be clusters of atypical keratinocytes invading the basement membrane to form small clusters in the papillary or reticular dermis.

Human papillomavirus-related Bowen disease

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Nonhealing, enlarging, hyperkeratotic papule that becomes inflamed and tender. May occur around the nail apparatus, as well as on other skin sites.

INVESTIGATIONS

Skin biopsy shows parakeratosis, atypical keratinocytes with or without koilocytes.

There may be clusters of atypical keratinocytes invading the basement membrane to form small clusters in the papillary or reticular dermis.

Lichen planus

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Usually widely spread and pruritic rash.

INVESTIGATIONS

Skin biopsy shows features characteristic of each lichen planus.

Epidermal nevus

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Pruritic plaques

INVESTIGATIONS

Skin biopsy shows features characteristic of each epidermal nevus.

Melanocytic nevus

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Generally brown or tan in color.

There are no tiny black dots on the surface.

INVESTIGATIONS

Skin biopsy shows features characteristic of melanocytic nevus.

Melanoma

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Lesions often have an irregular outline, diameter greater than 6 mm, and color variegation.

INVESTIGATIONS

Skin biopsy shows features characteristic of melanoma.

Molluscum contagiosum

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Lesions appear as umbilicated, pearl-like, smooth papules.

At least one third develop symptoms of local erythema, swelling, or pruritus.

Seen particularly in immunocompromised patients.

INVESTIGATIONS

Hematoxylin and eosin staining shows Henderson Patterson bodies, the definitive histological feature of molluscum contagiosum.

Tuberculosis verrucosa cutis

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Wart-like papule with a subtle inflammatory rim at trauma-prone sites, such as the hands and digits, as well as the lower extremities in children. Lesions enlarge gradually, often in a serpiginous manner, to form a reddish-brown verrucous plaque. Central areas may become fluctuant with pus and keratinaceous debris. After several years, the plaques may heal spontaneously.[4]

INVESTIGATIONS

Histology shows acute inflammation in the epidermis, pseudoepitheliomatous hyperplasia, microabcesses in the upper dermis, sparse granulomatous foci, and occasional bacilli.[4]

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