Differentials

Celiac disease

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Family history of celiac disease or personal history of other autoimmune diseases. Very similar presentation of nonspecific symptoms. Abdominal pain, nausea or vomiting, bloating, or excess intestinal gas common in celiac disease. Gluten intolerance is not present in abetalipoproteinemia.[4]

INVESTIGATIONS

Diagnosis is suggested by positive immunoglobulin A tissue transglutaminase serology, but must be confirmed by duodenal biopsy and histology.

Crohn disease

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Both patient groups present with a chronic course; however, diarrhea occurs with painful bouts in Crohn disease, whereas abetalipoproteinemia is painless. Additionally, patients with Crohn disease rarely present in infancy.

INVESTIGATIONS

Ileocolonoscopy with biopsies should be performed in the assessment of suspected Crohn disease.

Ulcerative colitis

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Both patient groups present with a chronic course; however, diarrhea occurs with painful bouts in ulcerative colitis, whereas abetalipoproteinemia is painless. Additionally, patients with ulcerative colitis disease rarely present in infancy.

INVESTIGATIONS

Colonoscopy with biopsy helps differentiate ulcerative colitis.

Viral gastroenteritis

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Abetalipoproteinemia is chronic while viral gastroenteritis is time-limited and occurs with fever.

INVESTIGATIONS

Usually a clinical diagnosis. Responds to oral rehydration therapy.

Child abuse

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Child abuse is generally manifested by faltering growth without steatorrhea or ocular signs and symptoms.

INVESTIGATIONS

In child abuse radiologic studies may show multiple healed fractures or suspicious spiral fractures.

Homozygotic hypobetalipoproteinemia (HHBL)

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Hepatic steatosis, while seen in both abetalipoproteinemia and HHBL, seems to be somewhat more prevalent in patients with HHBL.[2]

The heterozygous parent of a patient with HHBL typically has a reduced serum cholesterol concentration but is otherwise generally normal with no signs or symptoms.[7]

INVESTIGATIONS

In HHBL, the patient's parents will have low plasma LDL and low apolipoprotein B (apo B) levels.[7]

Genetic testing shows mutations in the apo B gene.[3]

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