History and exam

Key diagnostic factors

common

middle ear effusion

The presence of middle ear effusion without any signs of ear infection is a diagnostic criterion for OME.

On otoscopy, the tympanic membrane may appear dull, with golden fluid behind the drum. There may be bubbles or an air-fluid level.[2]​ The tympanic membrane may also be retracted. However, a normal looking tympanic membrane does not exclude OME, unless pneumatic otoscopy is normal as well.

no signs of acute infection

No signs of acute infection in the presence of middle ear effusion is a diagnostic criterion for OME.

Signs of acute infection (fever, ear pain, discharge from the ear, tympanic membrane bulging, and erythema) indicate acute otitis media rather than OME.

aural fullness or pressure

Approximately 13% of adults presenting with aural fullness are found to have OME.[60]​ This may be described as a feeling of ear blockage.

hearing loss

OME can be associated with a variable impact on hearing, from no hearing loss through to a moderate conductive hearing loss.[2]

Parents may notice signs of poor hearing, such as the child not responding when being called. Ask about indistinct speech, delayed language development or inattention. Infants and children may also present after a failed hearing screen or during the work up of speech delay.

Severe hearing loss may indicate pathology other than or in addition to OME and should be appropriately investigated.

Other diagnostic factors

common

slow progress within an education setting

A parent or caregiver may describe slow progress within an education setting, possibly due to hearing loss.[52]

failed hearing screen

Children may present with OME after failing a routine hearing screen through their school or primary care provider, or following a failed newborn hearing screen.

In one prospective study of 152 infants referred because of unilateral or bilateral failure of the universal newborn hearing screening test with an automated auditory brainstem response device, 84 (55.3%) had OME.[61]​ In infants with OME who fail a newborn hearing screen, follow-up is important to ensure that hearing is in fact normal once the OME resolves.[2]

speech delay

Chronic OME may be associated with speech delay.[2]

This may depend on the duration of OME as well as the extent of hearing loss during that time.[62]

signs of ear discomfort

Prelingual children may show signs of ear discomfort such as ear tugging or scratching the pinna.

uncommon

behavior problems

A parent or caregiver may describe behavior problems, possibly due to hearing loss.[52]

impaired gross motor skills and balance problems

Prelingual children may present with imbalance and impaired gross motor skills as OME can affect the peripheral vestibular system.[3][4]

Risk factors

strong

childhood

OME is common in childhood. It can be found in over 50% of children ages <1 year and 60% of children ages <2 years.[1][18]

One study using routine examination of the eardrum found OME in 15% to 40% of children ages between 1 and 5 years.[1][37]

In one study to identify the prevalence of OME in primary school children, a maximum prevalence of 12.9% was found in children ages 5 to 6 years.[1]​​[6]

In infants, the Eustachian tube is shorter and more horizontal than in older children and adults and is therefore less effective at clearing the contents of the middle ear.​[13][14]

Children have been found to be less able than adults to equalize negative middle ear pressure.[15]

upper respiratory tract infection

An upper respiratory tract infection is often the precipitating factor for OME.[2]​ Mucosal inflammation secondary to infection can lead to obstruction and dysfunction of the Eustachian tubes, thereby leading to fluid accumulation within the middle ear.[11]

acute otitis media

OME is typical following episodes of acute otitis media once the acute inflammation resolves.[38][39]

craniofacial anomalies

People with an unrepaired cleft palate almost universally experience chronic OME.[40]​ Abnormal muscular attachment of palatal muscles predisposes this population to Eustachian tube dysfunction and subsequent chronic OME.[41]​ After cleft palate repair, OME continues to be prevalent.[8]

Patients with trisomy 21 are also at increased risk of chronic OME. This may be secondary to poor active function of the Eustachian tubes.[11]

Eustachian tube dysfunction

The Eustachian tubes play an important role in maintaining the health and function of the middle ears. Specifically, the Eustachian tubes assist in pressure equalization of the middle ear and mucociliary clearance.[11] Dysfunction of the Eustachian tubes can therefore increase the risk of OME.[12]

genetic predisposition in children

Genes for immunoglobulin markers, cytokines, and mucin have been linked to an increased risk of OME.[16] Monozygotic twins have higher rates of concordant histories of otitis media compared with dizygotic twins.[17][18][19]

daycare attendance

Daycare attendance is a risk factor for recurrent OME (and acute otitis media).[33][10]​ This is likely related to increased exposure to viral pathogens, which cause upper respiratory tract infections.

adenoid hyperplasia, adenoiditis

Adenoid enlargement with obstruction of Eustachian tube orifices as well as the presence of microflora within the adenoid has been associated with OME.[2][27]

weak

allergic rhinitis

There is an association between allergy and OME, but there is currently no evidence of causation.[1]​ Children with OME have been found to have an increased prevalence of allergic rhinitis.[20]​ This may be secondary to allergy-related mucosal swelling and increased mucus production.[20]

Data suggest that age is an effect modifier of the association between allergic rhinitis and OME. Specifically, there is a significant association between allergic rhinitis and OME in children ages 6 years and older. The same association is not found in children younger than 6 years.[22]

environmental tobacco smoke

Tobacco smoking and exposure to second-hand tobacco smoke may increase the risk of otitis media with effusion. Proposed mechanisms include impaired mucociliary clearance of the Eustachian tubes and smoke-induced nasopharyngeal lymphoid hyperplasia.[23][24]​ In children, exposure to second-hand smoke is associated with higher odds of OME (and acute otitis media).[25]

sinonasal disease

Chronic rhinosinusitis with and without nasal polyposis has been associated with an increased risk of OME. Paranasal sinus disease was noted to be a main causative factor in 66% of 167 adults examined with OME.[24][26]

nasopharyngeal malignancy

The rate of nasopharyngeal carcinoma as the cause of adult-onset OME has been found to be approximately 5% to 6%.[28][24]​ The mechanism of OME may be due to Eustachian tube orifice obstruction or Eustachian tube dysfunction secondary to infiltration of tubal musculature.[29]​ Adults presenting with OME, particularly if unilateral and persistent, should be assessed for nasopharyngeal carcinoma via endoscopy.[28]

gastroesophageal reflux disease (GERD)

Symptoms of GERD have been found more frequently in people with OME compared to those without OME.[30]​ Pepsin and Helicobacter pylori have also been found in middle ear effusions.[31][1]​ While there is an association, a causal relationship has not been established.[1] Furthermore, treating GERD has not been associated with an increased rate of resolution of OME.[32]

low socioeconomic status

Low socioeconomic status has been associated with OME.[10]

male sex

Male sex has been associated with an increased incidence of persistent OME (and with acute otitis media).[10]​​[34]

Use of this content is subject to our disclaimer