Differentials

Leukemoid reaction

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

There are no specific clinical differentiating features.

INVESTIGATIONS

High WBC count resolves once underlying condition (e.g., infection) is treated.

Benign neutrophilic leukocytosis

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

There are no specific clinical differentiating features.

INVESTIGATIONS

Fluorescence in situ hybridization (FISH): negative for BCR::ABL1 fusion gene; absence of left-shifted WBC differential.

Atypical CML

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

There are no specific clinical differentiating features.

INVESTIGATIONS

Cytogenetics, FISH, quantitative polymerase chain reaction (qPCR): negative for BCR::ABL1 fusion gene; thrombocytopenia often prominent.

Chronic myelomonocytic leukemia

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

There are no specific clinical differentiating features.

INVESTIGATIONS

Bone marrow exam: dysplastic changes in the myeloid lineage.[38]

Peripheral blood smear: wide range of immature granulocytes and monocytosis.

FISH may rarely reveal a PDGFR gene rearrangement.[38]

Essential thrombocythemia

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

More indolent clinical course.

Unlikely to transform into acute leukemias.

INVESTIGATIONS

FISH: negative for BCR::ABL1, presence of JAK2, CALR, or MPL mutation.[39]

CBC: high platelet count.

Important to differentiate from early myelofibrosis.

Ph+ acute lymphoblastic leukemia

SIGNS / SYMPTOMS
INVESTIGATIONS
SIGNS / SYMPTOMS

Much more rapid disease course.

Patients often present with anemia and thrombocytopenia. B symptoms are common (e.g., fever, night sweats, and weight loss).

INVESTIGATIONS

Peripheral blood smear: lymphoid blasts.

Bone marrow exam and flow cytometry show terminal deoxynucleotidyl transferase-positive (TdT+) immature B lymphoblasts.

FISH: positive for 190-kDa BCR::ABL1 fusion.[1]

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