Treatment algorithm

Please note that formulations/routes and doses may differ between drug names and brands, drug formularies, or locations. Treatment recommendations are specific to patient groups: see disclaimer

INITIAL

suspected upper airway cough syndrome (UACS)

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1st line – 

empiric trial of therapy

Empiric therapy with a first-generation antihistamine (e.g., chlorpheniramine) plus a decongestant (e.g., pseudoephedrine) should be started when the etiology of cough is not apparent. Improvement or resolution of the cough in response to treatment is the pivotal factor in confirming a diagnosis of UACS.[1]​​

Open studies have shown therapeutic benefit with this combination regimen, and it is the recommended first-line treatment option.[1]​​

Approximately 60% of patients improved with this approach, and improvement is usually seen within 2 weeks.[1]​​[27]​​ Patients who have benefited from an empiric trial of this therapy should continue with it.

Pseudoephedrine is associated with risks of posterior reversible encephalopathy syndrome (PRES) and reversible cerebral vasoconstriction syndrome (RCVS); it should be avoided in patients with severe or uncontrolled hypertension or chronic or severe acute kidney disease.[28]

As sedation is a potential adverse effect of this regimen, it is recommended that treatment is commenced once daily in the evening prior to sleep for the first few days before increasing to the recommended dose.[1]​​

Primary options

chlorpheniramine: 4 mg orally (immediate-release) every 4-6 hours, maximum 24 mg/day

and

pseudoephedrine: 60 mg orally (immediate-release) every 4-6 hours, maximum 240 mg/day; 120 mg orally (extended-release) twice daily

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Plus – 

trigger identification and avoidance

Treatment recommended for ALL patients in selected patient group

Establishing the trigger and avoiding it is desirable but not always possible.

In patients with an allergic or environmental trigger, initiating an appropriate avoidance strategy is important. This can be difficult, as many people with rhinitis are sensitized to a perennial allergen.

Avoidance strategies include avoiding exposure, improved ventilation, filters, and personal protective devices (e.g., masks).[1]​​

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Consider – 

avoidance of nasal decongestant overuse

Treatment recommended for SOME patients in selected patient group

If rhinitis medicamentosa is suspected, it is important to discourage continuous use of topical nasal decongestants. If they have been used for some time, patients should gradually wean themselves off them (e.g., one nostril at a time).

ACUTE

confirmed upper airway cough syndrome (UACS)

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1st line – 

first-generation antihistamine + decongestant

Patients who have benefited from an empiric trial of this therapy should continue with it. Open studies have shown therapeutic benefit with this combination regimen, and it is the recommended first-line treatment option.[1]​​

Approximately 60% of patients improved with this approach, and improvement is usually seen within 2 weeks.[1]​​[27]​​

As sedation is a potential adverse effect of this regimen, it is recommended that treatment is commenced once daily in the evening prior to sleep for the first few days before increasing to the recommended dose.[1]​​

Pseudoephedrine is associated with risks of posterior reversible encephalopathy syndrome (PRES) and reversible cerebral vasoconstriction syndrome (RCVS); it should be avoided in patients with severe or uncontrolled hypertension or chronic or severe acute kidney disease.[28]

Primary options

chlorpheniramine: 4 mg orally (immediate-release) every 4-6 hours, maximum 24 mg/day

and

pseudoephedrine: 60 mg orally (immediate-release) every 4-6 hours, maximum 240 mg/day; 120 mg orally (extended-release) twice daily

Back
Consider – 

continued trigger avoidance

Treatment recommended for SOME patients in selected patient group

Establishing the trigger and avoiding it is desirable but not always possible.

In patients with an allergic or environmental trigger, initiating an appropriate avoidance strategy is important. This can be difficult, as many people with rhinitis are sensitized to a perennial allergen.

Avoidance strategies include avoiding exposure, improved ventilation, filters, and personal protective devices (e.g., masks).[1]​​

Back
Consider – 

avoidance of nasal decongestant overuse

Treatment recommended for SOME patients in selected patient group

If rhinitis medicamentosa is suspected, it is important to discourage continuous use of topical nasal decongestants. If they have been used for some time, patients should gradually wean themselves off them (e.g., one nostril at a time).

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Consider – 

specialist management of structural upper airway abnormalities

Treatment recommended for SOME patients in selected patient group

Management of structural upper airway abnormalities should be guided by an ENT specialist. For example, significant nasal septal deviation may require surgical correction.

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Consider – 

speech and language therapy

Treatment recommended for SOME patients in selected patient group

There is some evidence for the use of nonpharmacologic interventions such as speech and language therapy in the management of chronic cough with upper airway features.[26][32]​​[33][34][35]

One randomized, placebo-controlled trial in patients with chronic cough demonstrated that 4 treatment sessions significantly improved cough and upper airway scores.[33]

Treatment modalities include educational information, strategies to reduce cough, improved laryngeal hygiene (e.g., increased hydration), and psychoeducational counseling.

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Plus – 

intranasal corticosteroid, antihistamine, cromolyn, or leukotriene receptor antagonist

Treatment recommended for ALL patients in selected patient group

First-line treatments for rhinitis include intranasal corticosteroids (e.g., mometasone), antihistamines (e.g., azelastine), or cromolyn sodium.[1]​​

Leukotriene receptor antagonists (e.g., montelukast) also appear to improve symptoms in patients with allergic rhinitis.[1] The Food and Drug Administration (FDA) has strengthened its warnings for montelukast about the risk of serious behavior and mood-related changes. For allergic rhinitis, the FDA has determined that montelukast should be reserved for those who are not treated effectively with or cannot tolerate other allergy medicines.[30]

Primary options

mometasone nasal: 100 micrograms (2 sprays) in each nostril once daily

OR

azelastine nasal: 137-274 micrograms (1-2 sprays) in each nostril twice daily

OR

cromolyn: 5.2 mg (1 spray) into both nostrils four to six times daily

Secondary options

montelukast: 10 mg orally once daily

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Plus – 

antibiotic or antifungal

Treatment recommended for ALL patients in selected patient group

Excess sputum production may indicate bacterial or fungal sinusitis.

Common pathogens include Streptococcus pneumoniae and Haemophilus influenzae

Appropriate antibiotic therapy (or antifungal therapy depending on the etiology) should be initiated after CT imaging and an ENT assessment if warranted.[29]

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Plus – 

proton-pump inhibitor

Treatment recommended for ALL patients in selected patient group

Treatment of co-existing gastroesophageal reflux is an important consideration.

One study found that treatment with a proton-pump inhibitor (rabeprazole) for 90 days reduced rhinitis symptom scores and chronic cough.[31]

Omeprazole or lansoprazole may also be used.

Primary options

rabeprazole: 20-40 mg/day orally

OR

omeprazole: 20-40 mg/day orally

OR

lansoprazole: 15-30 mg/day orally

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Consider – 

sinus imaging

Treatment recommended for SOME patients in selected patient group

Patients who do not respond to targeted treatment with a first-generation antihistamine plus decongestant should undergo sinus imaging, as chronic sinusitis may be clinically silent.

If the diagnosis is confirmed, additional targeted treatment for chronic sinusitis is recommended (e.g., antibiotics, intranasal corticosteroids).

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Please note that formulations/routes and doses may differ between drug names and brands, drug formularies, or locations. Treatment recommendations are specific to patient groups. See disclaimer

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