History and exam

Key diagnostic factors

common

headache

Typically worse in morning and gets better during the day (sign of elevated intracranial pressure).[24]

Often relieved by vomiting.

nausea and vomiting

Typically worse in morning and gets better during the day (sign of elevated intracranial pressure).[24]

Vomiting occurs in the absence of fever or other signs of an infection, and often relieves the headache.[24]

Other diagnostic factors

common

behavioural changes

Very young children may not be able to express how they are feeling, so it will often be the parents/carers who describe changes in their child's behaviour (e.g., irritability, decreased social interaction).

diplopia

Due to sixth cranial nerve weakness.

ophthalmoplegias

Difficulty in lateral gaze may occur from unilateral or bilateral sixth cranial nerve weakness.

bulging anterior fontanelle

Common in children <2 years of age.

Sign of elevated intracranial pressure due to hydrocephalus in an infant, or tumour if >6 months.[24]

papilloedema

Secondary to elevated intracranial pressure.

uncommon

nystagmus

Often occurs late in the presentation or not at all.

head tilt

Usually occurs late in the presentation or not at all.

ataxia

Includes balance difficulty or hand clumsiness.

Usually occurs late in the presentation or not at all.

dysmetria

Usually occurs late in the presentation or not at all.

lethargy

Due to obstructive hydrocephaly. Usually occurs late in the presentation or not at all.

Risk factors

weak

age 1 to 9 years

Peak age of presentation is between 1 and 9 years; however, presentation can occur at any age.[5]​​

male sex

There is a slight male predominance.[5]

associated inherited familial syndromes

There are reported associations with the following diseases: ataxia-telangiectasia, Rubinstein-Taybi syndrome, Gorlin syndrome (nevoid basal cell carcinoma syndrome), Turcot syndrome, Lynch syndrome/constitutional mismatch repair deficiency, Li-Fraumeni syndrome (TP53 germline mutation), and other germline mutations (BRCA2, PALB2, GPR161, ELP1, CREBBP, and EP300).[13][14][15]​​

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