History and exam

Key diagnostic factors

common

presence of risk factors

Key risk factors include smoking, moderate- to high-level alcohol consumption, previous subarachnoid haemorrhage (SAH), family history of SAH, and heritable connective tissue diseases.

headache

New, never-before-experienced headache suggests ruptured cerebral aneurysm.

The character of the headache can be quite variable.

Other diagnostic factors

uncommon

seizures

New, never-before-experienced focal or generalised seizures.

nuchal rigidity

New neck stiffness that includes pain on stretching the neck meninges with manoeuvres. More commonly found in subarachnoid haemorrhage.

decreased level of consciousness

More commonly found in subarachnoid haemorrhage. Usually related to concurrent hydrocephalus.

focal neurological deficit

Variable, depending on mass effect of aneurysm and/or haematoma. More commonly found in subarachnoid haemorrhage.

The classic syndrome is that of a third nerve palsy with pupillary dysfunction from a posterior communicating artery aneurysm exerting mass effect.[24]

Risk factors

strong

smoking

Cigarette smoking is an independent risk factor for aneurysm formation, growth, and rupture.[12][15]

moderate- to high-level alcohol consumption

Moderate- to high-level alcohol consumption is an independent risk factor for aneurysmal subarachnoid haemorrhage.[12]

family history of subarachnoid haemorrhage

Highest in siblings. No single Mendelian model but several possible patterns of inheritance identified. Most likely autosomal dominant.[8]

previous subarachnoid haemorrhage

Multiple aneurysms are consistently seen in follow-up studies of patients with subarachnoid aneurysmal haemorrhage (SAH). One cohort study followed up 610 patients who had previous SAH and detected aneurysms in 96 (16%) patients, after a mean interval of 8.9 years.[16]

heritable connective tissue disease

Considered specifically in patients with autosomal dominant polycystic kidney disease, Ehlers-Danlos syndrome type IV, neurofibromatosis type 1, and Marfan's syndrome.[8]

weak

hypertension

Hypertension is a weak risk factor for aneurysm formation and rupture, although population studies examining its importance have produced conflicting results.

head trauma

May cause damage to the vascular wall leading to aneurysm formation.

intracranial infection

May cause damage to the vascular wall leading to aneurysm formation.

tumour

The presence of an intracranial tumour can lead to aneurysm formation.

arteriovenous malformations or fistulas

Arteriovenous malformations or fistulas in the intracranial circulation can develop into aneurysms.

drug abuse

Drug abuse can lead to cerebral aneurysm formation (particularly amphetamines, cocaine, and ecstasy).[17]

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