History and exam
Key diagnostic factors
common
presence of risk factors
Key risk factors include: exposure to TB; born in Asia, Latin America, or Africa; HIV infection; immunosuppressive medicines; haematological or head/neck malignancy; ESRD; apical fibrosis; and very young age.
enlarged lymph node
Painless and gradual enlargement of unilateral or bilateral cervical or supraclavicular nodes over a period of weeks; nodes are typically firm.
Other presentations include painful or fluctuant nodes that may have drainage. Nodes may rarely be located in axillary or inguinal regions.[74][75]
Concomitant pulmonary TB may also be seen.
pleuritic chest pain
Present in up to 75% of patients with pleural TB.[75]
skeletal pain
Pain is common in skeletal TB and its location depends on the site of involvement.
Pain may evolve over weeks to months.
In Pott's disease, kyphosis may be present and focal tenderness may be present.
If the hip or knee is involved, the patient may complain of pain with walking and local swelling may be present.
If untreated, cold abscesses may form that are not tender or erythematous and are more common in HIV-positive patients. If these rupture, a draining sinus tract forms.[52]
urinary symptoms
Seen in GU TB. Includes dysuria, haematuria, and urinary frequency.
abdominal swelling
In peritoneal TB, swelling seen in over 90% of patients.
The classic doughy abdomen is associated with the chronic fibroadhesive form and is rarely seen.
abdominal pain
Diffuse pain may be seen in 75% of patients with TB peritonitis.
In patients with TB enteritis, pain is present in 80% to 90%, most commonly in the right lower quadrant (RLQ). A palpable mass may be present.
uncommon
headache
Seen in TB meningitis.
Other diagnostic factors
uncommon
cough
altered mental status
Seen in TB meningitis.
neurological symptoms
Cranial nerve involvement results from TB meningitis, as the process is located primarily at the base of the brain.
Peripheral nerve symptoms may result from vertebral involvement with cord compression. May include numbness, weakness, or paralysis. Vertebrae in thoracolumbar region most commonly involved.
hepatomegaly
May be seen in up to one-third of patients with disseminated TB. May also have splenomegaly.[16]
abnormal chest examination
Chest examination may be abnormal if pulmonary TB also present or if pleural disease.
Possible findings include a friction rub, crackles, decreased breath sounds, or dullness to percussion.
fever
weight loss of more than 10% body weight
night sweats
If present, usually drenching. Common in disseminated TB.[78]
dyspnoea
May be seen in disseminated TB.
asymptomatic
Particularly patients with GU TB, who may be suspected on routine urinalysis.
erythema nodosum and erythema induratum
Painful raised erythematous nodules over pretibial region or on the calves.
Risk factors
strong
exposure to TB
Exposure to an infectious case (i.e., pulmonary or laryngeal TB) is necessary but not sufficient for development of TB. Among household contacts, about one third will acquire latent TB infection and 1% to 2% will have active TB disease. Persons with recently acquired infection (e.g., new TB skin test conversion) have an increased risk of developing active TB, although this relationship holds less strong for EPTB compared with pulmonary TB.[20][21]
born in Asia, Latin America, or Africa
HIV infection
HIV infection increases the risk for both progression to primary disease and reactivation of latent disease. The risk for reactivation in an HIV-positive patient with latent TB infection is up to 10% per year, as opposed to a 10% lifetime risk in HIV-negative people. Extrapulmonary manifestations of TB are more common in HIV, and patients are at a higher risk for central nervous system TB.[3][24][25][26][27]
immunosuppressive medicines
Especially systemic corticosteroids and tumour necrosis factor (TNF) antagonists. Risk with corticosteroids increases with increasing doses (odds ratio 7.7 for >5 mg per day of prednisolone) and varies with underlying condition. Patients receiving TNF antagonists are at 2-20 times higher risk for TB; more than 50% of TNF antagonist-related TB cases will be extrapulmonary.[28] The risk for TB with infliximab is greater than etanercept. Relative risk following organ transplantation is 20- to 74-fold higher.[29][30]
haematological or head/neck malignancy
Patients with haematological malignancy and head-and-neck cancer have a higher risk than people without malignancies.[31] The risk with other types of cancer has not been determined.
end stage renal disease
Patients on haemodialysis are at increased risk of EPTB. Patients on peritoneal dialysis are at increased risk for peritoneal TB.[15]
apical fibrosis
Patients whose chest x-ray shows fibrotic changes consistent with prior pulmonary TB are at higher risk for developing active disease again (estimated risk 0.3% per year).[32]
weak
intravenous drug use
Even without HIV infection.[33]
female sex
Odds ratio for development of EPTB compared with pulmonary TB is 3.69.[20]
Asian, black, and Native American ethnicity
EPTB is more likely in Asian, black, and Native American people than in white people.[34]
malnutrition
Includes people with low body weight (<90% of ideal body weight), coeliac disease, and history of gastrectomy. Risk higher in patients who have undergone jejunoileal bypass.
alcoholism
Hard to separate from other risk factors.
diabetes
A relative risk of 2 to 4 if uncontrolled.
cirrhosis
Increased risk for peritoneal TB.[15]
high-risk congregate settings
Resident or employee of correctional facility, homeless shelter, or nursing home.
low socioeconomic status
Multivariate models suggest at least half the risk attributed to ethnicity (black, Hispanic, Native Americans) may be due to low socioeconomic status.[35]
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