History and exam
Key diagnostic factors
common
malpositioned or absent testis
Affected testis may be palpable or nonpalpable, unilateral, or bilateral.
palpable cryptorchid testis
Unable to be manipulated into the scrotum or immediately returns to a higher position after being manipulated into the scrotum.
nonpalpable testis
Testis is unable to be located/palpated in the scrotum, inguinal canal, or femoral or perineal regions despite thorough physical exam.
Other diagnostic factors
common
testicular asymmetry
scrotal hypoplasia or asymmetry
retractile testis
Testis that is located in a suprascrotal position but can be manipulated down without pain into the scrotum and remains there after traction is released.
uncommon
ascending cryptorchidism
Cryptorchidism may also present as an ascending event in patients with previously documented normal testis position in the scrotum, later noted to have ascended into the high portion of the scrotum or the inguinal canal.
hypospadias
Presence of hypospadias should prompt referral for genetic testing and further urologic assessment.
micropenis
Penile length less than 2 standard deviations below normal for age.
secondary sex characteristics/pubertal signs in prepubertal/pubertal patients
These should be sought for a patient presenting with cryptorchidism at an older age.
surgical scar in the inguinal region
Cryptorchidism may be iatrogenic.
Risk factors
strong
family history of cryptorchidism
prematurity
The descent of the testis from the inguinal region into the scrotum generally occurs during 24 to 35 weeks of gestation.[37] Premature infants have a 9% to 45% incidence of cryptorchidism at birth and an odds ratio (OR) of 2.5 compared with controls.[4][38][39] Cryptorchidism at birth may resolve in many of these infants when adjusted for gestational age.
low birth weight (<2.5 kg) and/or small for gestational age
Case control studies have demonstrated a separate increased risk of cryptorchidism in infants small for gestational age and/or low birth weight even when controlling for prematurity, with OR ranging from 1.2 to 3.53 and a prevalence of 1.0 to 4.6%.[4][39][40] A study has shown that those infants who reach normal weight by 1 year of age have a higher chance of testicular descent than those who remain underweight.[41]
weak
environmental exposures
There is growing appreciation of the toxicity of environmental endocrine disruptors.[42] These may include organochlorines such as polychlorinated biphenyls (PCBs), environmental estrogens, phthalate esters, and bisphenol-A (BPA).[15][43][44][45][46] A 13% nationwide increase in cryptorchidism was observed following the Fukushima nuclear accident in Japan in 2011.[47] Analgesic consumption, smoking, and pesticides or agricultural exposure have also all been linked to increased risk of cryptorchidism, although a review of literature demonstrates that studies are often conflicting and difficult to interpret due to the complexity of chemical compound mixtures and the unclear pathophysiology of cryptorchidism.[5][8][12][16][17][18][19][20][21][48][49][50]
maternal alcohol use
gestational diabetes
Abnormalities of maternal glucose metabolism during pregnancy were associated with an OR of 2.44 to 3.98 increased risk of cryptorchidism in Finnish newborn boys.[51]
prior inguinal surgery
Cryptorchidism may be iatrogenic in such cases.[52]
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