Differentials
Neurofibromatosis type 2 (NF2)
SIGNS / SYMPTOMS
Bilateral vestibular schwannomas, intracranial meningiomas and/or ependymomas, spinal glioma, paraspinal schwannomas, cutaneous schwannomas, posterior subcapsular cataracts, pigmentary retinopathy, absence of typical café au lait spots.[26]
INVESTIGATIONS
Identification of a mutation at the NF2 locus in chromosome band 22q11.[27]
McCune-Albright syndrome
SIGNS / SYMPTOMS
Very few or no café au lait spots, no axillary freckling, polyostotic fibrous dysplasia.[28]
INVESTIGATIONS
Identification of an activating mutation in the GNAS1 gene locus in chromosome band 20q13.2.
Familial café au lait spots
NF1-like syndrome (Legius syndrome)
SIGNS / SYMPTOMS
Absence of neurofibromas or optic pathway glioma or other features of NF1 except for the presence of café au lait spots, axillary freckling, macrocephaly, facial dysmorphism, and perhaps learning difficulties.[30]
INVESTIGATIONS
Identification of a mutation in the SPRED1 gene in chromosome band 15q13.2.[31]
Proteus syndrome
Age-related cutaneous neurofibromas
SIGNS / SYMPTOMS
One to several cutaneous neurofibromas that do not buttonhole (i.e., push deeper into the dermis when pressed) and appear only in or beyond the fifth decade of life.[9]
INVESTIGATIONS
Absence of an NF1 mutation in a non-neurofibroma specimen.
Isolated diffuse plexiform neurofibroma
SIGNS / SYMPTOMS
A diffuse plexiform neurofibroma in the absence of any other features of NF1.[9]
INVESTIGATIONS
Absence of an NF1 mutation in a non-neurofibroma specimen.
Schwannomatosis
SIGNS / SYMPTOMS
Little or no skin pigmentation such as café au lait spots or intertriginous freckling. Multiple schwannomas.[34]
INVESTIGATIONS
Tumor biopsy uniformly "schwannoma". Absence of splicing or missense mutations.
Congenital mismatch repair deficiency
SIGNS / SYMPTOMS
May have six or more café au lait spots, with typically more ragged edges.
INVESTIGATIONS
Homozygous mutations in one of the MMR genes: PMS2, MLH1, MSH6, MSH2.
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