High-flow oxygen therapy, hyperbaric oxygen, and supportive therapy are the key treatments for CO poisoning. High-flow oxygen therapy should be initiated as soon as the diagnosis is suspected, and should not be discontinued until the diagnosis is ruled out. It is recommended that persistently symptomatic patients are treated with hyperbaric oxygen and re-treated up to a maximum of three times.[2]Hampson NB, Piantadosi CA, Thom SR, et al. Practice recommendations in the diagnosis, management, and prevention of carbon monoxide poisoning. Am J Respir Crit Care Med. 2012 Dec 1;186(11):1095-101.
http://www.atsjournals.org/doi/full/10.1164/rccm.201207-1284CI#.VXHCJ8-jMyo
http://www.ncbi.nlm.nih.gov/pubmed/23087025?tool=bestpractice.com
Depending on where and how the CO poisoning occurred, patients may have multiple other injuries to treat, such as burns, smoke inhalation injury, or overdoses. Contact the Poison Center early for advice on management.
America's Poison Centers
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Supportive care and monitoring
Remove patient(s) from the CO exposure environment immediately. Resuscitate the patient according to standard guidelines, as needed.[53]Panchal AR, Bartos JA, Cabañas JG, et al. Part 3: adult basic and advanced life support: 2020 American Heart Association guidelines for cardiopulmonary resuscitation and emergency cardiovascular care. Circulation. 2020 Oct 20;142(16_suppl_2):S366-468.
https://www.doi.org/10.1161/CIR.0000000000000916
http://www.ncbi.nlm.nih.gov/pubmed/33081529?tool=bestpractice.com
Patients with CO poisoning can be critically ill, and all patients should have close cardiopulmonary and neurologic monitoring throughout treatment, with close attention to fluid balance particularly for hypotensive patients. See Shock.
Patients with altered consciousness should be considered for endotracheal intubation. Seizures and cardiac complications should be treated urgently.
Carboxyhemoglobin (CO-Hb) can be monitored with blood gas CO-oximetry, although levels may be low in patients receiving oxygen for a period of time, or with significant delays to presentation. An ECG and cardiac biomarkers may be monitored to detect evidence of cardiac dysfunction in moderately or severely poisoned patients (based on symptom severity and/or CO-Hb levels of 25% or more), and in those with suspected cardiac involvement.[30]Wolf SJ, Maloney GE, Shih RD, et al; American College of Emergency Physicians Clinical Policies Subcommittee (Writing Committee) on Carbon Monoxide Poisoning. Clinical policy: critical issues in the evaluation and management of adult patients presenting to the emergency department with acute carbon monoxide poisoning. Ann Emerg Med. 2017 Jan;69(1):98-107.e6.
http://www.annemergmed.com/article/S0196-0644(16)31345-2/fulltext
http://www.ncbi.nlm.nih.gov/pubmed/27993310?tool=bestpractice.com
Also consider monitoring lactate to estimate metabolic acidosis, blood glucose to eliminate hypoglycemia as a cause for altered mental status and to check for hyperglycemia, and creatine kinase to monitor for rhabdomyolysis. Fetal monitoring is necessary in pregnant women to detect any evidence of fetal distress or fetal compromise.[16]Kao LW, Nanagas KA. Toxicity associated with carbon monoxide. Clin Lab Med. 2006 Mar;26(1):99-125.
http://www.ncbi.nlm.nih.gov/pubmed/16567227?tool=bestpractice.com
Those at highest risk for death are patients with severe metabolic acidosis (low pH on blood gas), those with very high CO-Hb levels (>25%), fire as a source of CO exposure, loss of consciousness or need for mechanical ventilation.[54]Hampson NB, Hauff NM. Risk factors for short-term mortality from carbon monoxide poisoning treated with hyperbaric oxygen. Crit Care Med. 2008 Sep;36(9):2523-7.
http://www.ncbi.nlm.nih.gov/pubmed/18679118?tool=bestpractice.com
Oxygen
Supplemental 100% oxygen through a non-rebreather mask or high-flow nasal cannula should be given to all patients with CO poisoning regardless of CO-oximetry readings or SaO2 levels (SaO2 alone is not helpful in detecting CO poisoning because it does not distinguish CO-Hb from oxyhemoglobin).[55]Yesiloglu O, Gulen M, Satar S, et al. Treatment of carbon monoxide poisoning: high-flow nasal cannula versus non-rebreather face mask. Clin Toxicol (Phila). 2021 May;59(5):386-91.
http://www.ncbi.nlm.nih.gov/pubmed/32959716?tool=bestpractice.com
Supplemental oxygen reduces hypoxia and increased PaO2 decreases the half-life of CO-Hb through facilitating the elimination of CO in the lungs. Although oxygen therapy is given regardless of arterial PaO2 levels, measuring arterial blood gas can assess acid/base status. Low pH is associated with mortality. Severe acidosis could be considered an indication for hyperbaric oxygen.[16]Kao LW, Nanagas KA. Toxicity associated with carbon monoxide. Clin Lab Med. 2006 Mar;26(1):99-125.
http://www.ncbi.nlm.nih.gov/pubmed/16567227?tool=bestpractice.com
See Evaluation of metabolic acidosis.
Administering 100% oxygen reduces the half life of carbon monoxide to 85 minutes compared to 4-5 hours in standard room air conditions.[30]Wolf SJ, Maloney GE, Shih RD, et al; American College of Emergency Physicians Clinical Policies Subcommittee (Writing Committee) on Carbon Monoxide Poisoning. Clinical policy: critical issues in the evaluation and management of adult patients presenting to the emergency department with acute carbon monoxide poisoning. Ann Emerg Med. 2017 Jan;69(1):98-107.e6.
http://www.annemergmed.com/article/S0196-0644(16)31345-2/fulltext
http://www.ncbi.nlm.nih.gov/pubmed/27993310?tool=bestpractice.com
This can be further reduced to around 20 minutes with hyperbaric oxygen.[30]Wolf SJ, Maloney GE, Shih RD, et al; American College of Emergency Physicians Clinical Policies Subcommittee (Writing Committee) on Carbon Monoxide Poisoning. Clinical policy: critical issues in the evaluation and management of adult patients presenting to the emergency department with acute carbon monoxide poisoning. Ann Emerg Med. 2017 Jan;69(1):98-107.e6.
http://www.annemergmed.com/article/S0196-0644(16)31345-2/fulltext
http://www.ncbi.nlm.nih.gov/pubmed/27993310?tool=bestpractice.com
Hyperbaric oxygen therapy
Hyperbaric oxygen is 100% oxygen in a pressurized chamber. It is used to enhance clearance of CO-Hb from the body through increasing the PaO2 in the lungs. The therapeutic goal of treatment with hyperbaric oxygen is the prevention of long-term and permanent neurocognitive dysfunction, more than the enhancement of short-term survival rates.[2]Hampson NB, Piantadosi CA, Thom SR, et al. Practice recommendations in the diagnosis, management, and prevention of carbon monoxide poisoning. Am J Respir Crit Care Med. 2012 Dec 1;186(11):1095-101.
http://www.atsjournals.org/doi/full/10.1164/rccm.201207-1284CI#.VXHCJ8-jMyo
http://www.ncbi.nlm.nih.gov/pubmed/23087025?tool=bestpractice.com
Hyperbaric oxygen therapy should be considered in patients with: loss of consciousness, ischemic cardiac changes, neurologic deficits, significant metabolic acidosis, or peak CO-Hb levels ≥25%.[4]Rose JJ, Wang L, Xu Q, et al. Carbon monoxide poisoning: pathogenesis, management, and future directions of therapy. Am J Respir Crit Care Med. 2017 Mar 1;195(5):596-606.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5363978
http://www.ncbi.nlm.nih.gov/pubmed/27753502?tool=bestpractice.com
[27]Centers for Disease Control and Prevention. Clinical guidance for carbon monoxide poisoning following disasters and severe weather. Jul 2024 [internet publication].
https://www.cdc.gov/carbon-monoxide/hcp/clinical-guidance
[56]Hampson NB, Moon RE, Weaver LK. Another perspective on ACEP policy on critical issues in carbon monoxide poisoning: Invited commentary. Undersea Hyperb Med. 2017 Mar-Apr;44(2):89-92.
http://www.ncbi.nlm.nih.gov/pubmed/28777898?tool=bestpractice.com
[57]Weaver LK, Hopkins RO, Chan KJ, et al. Hyperbaric oxygen for acute carbon monoxide poisoning. N Engl J Med. 2002 Oct 3;347(14):1057-67.
http://www.nejm.org/doi/full/10.1056/NEJMoa013121#t=article
http://www.ncbi.nlm.nih.gov/pubmed/12362006?tool=bestpractice.com
[58]Cowl CT. Justifying hyperbaric oxygen delivery for carbon monoxide poisoning: time to respond to pressure with a large-scale randomized controlled trial. Chest. 2017 Nov;152(5):911-13.
https://journal.chestnet.org/article/S0012-3692(17)31318-1/pdf
http://www.ncbi.nlm.nih.gov/pubmed/29126532?tool=bestpractice.com
[59]Weaver LK. Carbon monoxide poisoning. Undersea Hyperb Med. 2020 First Quarter;47(1):151-69.
https://www.doi.org/10.22462/01.03.2020.17
http://www.ncbi.nlm.nih.gov/pubmed/32176957?tool=bestpractice.com
The American College of Emergency Physicians (ACEP) recommends that emergency physicians use either hyperbaric oxygen therapy or high-flow normobaric therapy for acute CO poisoning. The ACEP acknowledges that there is no clear evidence that one approach is more effective than the other in terms of preventing delayed neurocognitive sequelae. The ACEP’s recommendation is intended to support physicians who choose not to refer patients for hyperbaric oxygen therapy owing to, for example, time or geographic constraints.[30]Wolf SJ, Maloney GE, Shih RD, et al; American College of Emergency Physicians Clinical Policies Subcommittee (Writing Committee) on Carbon Monoxide Poisoning. Clinical policy: critical issues in the evaluation and management of adult patients presenting to the emergency department with acute carbon monoxide poisoning. Ann Emerg Med. 2017 Jan;69(1):98-107.e6.
http://www.annemergmed.com/article/S0196-0644(16)31345-2/fulltext
http://www.ncbi.nlm.nih.gov/pubmed/27993310?tool=bestpractice.com
One systematic review of six clinical trials found no significant benefit to hyperbaric oxygen therapy in preventing delayed neurocognitive deficits.[60]Buckley NA, Juurlink DN, Isbister G, et al. Hyperbaric oxygen for carbon monoxide poisoning. Cochrane Database Syst Rev. 2011 Apr 13;2011(4):CD002041.
https://www.cochranelibrary.com/cdsr/doi/10.1002/14651858.CD002041.pub3/full
http://www.ncbi.nlm.nih.gov/pubmed/21491385?tool=bestpractice.com
A key limitation of this analysis was the heterogeneity in the trials on primary outcome, the dosage and number of hyperbaric oxygen therapy sessions, and delays to therapy. Negative clinical trials typically used a clinically ineffective dose of hyperbaric oxygen.[59]Weaver LK. Carbon monoxide poisoning. Undersea Hyperb Med. 2020 First Quarter;47(1):151-69.
https://www.doi.org/10.22462/01.03.2020.17
http://www.ncbi.nlm.nih.gov/pubmed/32176957?tool=bestpractice.com
The only included randomized controlled trial (RCT) that met all Consolidated Standards of Reporting Trials (CONSORT) criteria for assessing quality of RCTs showed a significant benefit from hyperbaric oxygen.[57]Weaver LK, Hopkins RO, Chan KJ, et al. Hyperbaric oxygen for acute carbon monoxide poisoning. N Engl J Med. 2002 Oct 3;347(14):1057-67.
http://www.nejm.org/doi/full/10.1056/NEJMoa013121#t=article
http://www.ncbi.nlm.nih.gov/pubmed/12362006?tool=bestpractice.com
The study used the following protocol:
First session: 3.0 atmospheres for 60 minutes, followed by 2.0 atmospheres for 60 minutes
Second and third sessions: 2.0 atmospheres for 120 minutes.
One subsequent consensus opinion recommended the use of this trial to guide clinical practice until further information from future studies becomes available.[2]Hampson NB, Piantadosi CA, Thom SR, et al. Practice recommendations in the diagnosis, management, and prevention of carbon monoxide poisoning. Am J Respir Crit Care Med. 2012 Dec 1;186(11):1095-101.
http://www.atsjournals.org/doi/full/10.1164/rccm.201207-1284CI#.VXHCJ8-jMyo
http://www.ncbi.nlm.nih.gov/pubmed/23087025?tool=bestpractice.com
[4]Rose JJ, Wang L, Xu Q, et al. Carbon monoxide poisoning: pathogenesis, management, and future directions of therapy. Am J Respir Crit Care Med. 2017 Mar 1;195(5):596-606.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5363978
http://www.ncbi.nlm.nih.gov/pubmed/27753502?tool=bestpractice.com
[56]Hampson NB, Moon RE, Weaver LK. Another perspective on ACEP policy on critical issues in carbon monoxide poisoning: Invited commentary. Undersea Hyperb Med. 2017 Mar-Apr;44(2):89-92.
http://www.ncbi.nlm.nih.gov/pubmed/28777898?tool=bestpractice.com
[58]Cowl CT. Justifying hyperbaric oxygen delivery for carbon monoxide poisoning: time to respond to pressure with a large-scale randomized controlled trial. Chest. 2017 Nov;152(5):911-13.
https://journal.chestnet.org/article/S0012-3692(17)31318-1/pdf
http://www.ncbi.nlm.nih.gov/pubmed/29126532?tool=bestpractice.com
Providers should take this ongoing uncertainty into account when considering hyperbaric oxygen therapy in patients with CO poisoning. Complications from hyperbaric oxygen therapy include pulmonary and ear barotrauma, and seizures.[57]Weaver LK, Hopkins RO, Chan KJ, et al. Hyperbaric oxygen for acute carbon monoxide poisoning. N Engl J Med. 2002 Oct 3;347(14):1057-67.
http://www.nejm.org/doi/full/10.1056/NEJMoa013121#t=article
http://www.ncbi.nlm.nih.gov/pubmed/12362006?tool=bestpractice.com
If hyperbaric oxygen is required, some centers recommend that it should be started within 6 hours and no later than 24 hours.[40]Eichhorn L, Thudium M, Jüttner B. The diagnosis and treatment of carbon monoxide poisoning. Dtsch Arztebl Int. 2018 Dec 24;115(51-52):863-70.
https://www.aerzteblatt.de/int/archive/article/203936
http://www.ncbi.nlm.nih.gov/pubmed/30765023?tool=bestpractice.com
For nonintubated patients, hyperbaric treatments are recommended until signs and symptoms have resolved, or until a clinical plateau has been reached (usually a maximum of three treatments).[59]Weaver LK. Carbon monoxide poisoning. Undersea Hyperb Med. 2020 First Quarter;47(1):151-69.
https://www.doi.org/10.22462/01.03.2020.17
http://www.ncbi.nlm.nih.gov/pubmed/32176957?tool=bestpractice.com
For intubated patients whose neurologic exam must be abbreviated out of necessity, three treatments are recommended.[61]Weaver LK, Deru K, Churchill S, et al. A randomized trial of one versus three hyperbaric oxygen sessions for acute carbon monoxide poisoning. Undersea Hyperb Med. 2023 Third Quarter;50(3):325-42.
https://www.semanticscholar.org/paper/A-randomized-trial-of-one-versus-three-hyperbaric-Weaver-Deru/d5070f1e28a9972d3d5092e143ac691aa33a843e
http://www.ncbi.nlm.nih.gov/pubmed/37708067?tool=bestpractice.com
Treat hyperglycemia
Hyperglycemia can occur after various types of severe brain injury, regardless of the presence of preexisting diabetes mellitus.[62]Garg R, Chaudhuri A, Munschauer F, et al. Hyperglycemia, insulin, and acute ischemic stroke: a mechanistic justification for a trial of insulin infusion therapy. Stroke. 2006 Jan;37(1):267-73.
https://www.doi.org/10.1161/01.STR.0000195175.29487.30
http://www.ncbi.nlm.nih.gov/pubmed/16306459?tool=bestpractice.com
[63]Shi J, Dong B, Mao Y, et al. Review: traumatic brain injury and hyperglycemia, a potentially modifiable risk factor. Oncotarget. 2016 Oct 25;7(43):71052-61.
https://www.doi.org/10.18632/oncotarget.11958
http://www.ncbi.nlm.nih.gov/pubmed/27626493?tool=bestpractice.com
[64]Chen S, Liu Z. Effect of hyperglycemia on all-cause mortality from pediatric brain injury: a systematic review and meta-analysis. Medicine (Baltimore). 2020 Nov 25;99(48):e23307.
https://www.doi.org/10.1097/MD.0000000000023307
http://www.ncbi.nlm.nih.gov/pubmed/33235087?tool=bestpractice.com
There is evidence that neurologic outcomes are worse in patients with hyperglycemia, although evidence specific to CO poisoning is limited.[33]White SR, Penney DG. Initial study: effects of insulin and glucose treatment on neurologic outcome after CO poisoning. Ann Emerg Med. 1994;23:606-7.[65]Santana D, Mosteiro A, Pedrosa L, et al. Clinical relevance of glucose metrics during the early brain injury period after aneurysmal subarachnoid hemorrhage: an opportunity for continuous glucose monitoring. Front Neurol. 2022 Sep 12:13:977307.
https://www.frontiersin.org/articles/10.3389/fneur.2022.977307/full
http://www.ncbi.nlm.nih.gov/pubmed/36172028?tool=bestpractice.com
[66]Peffer J, McLaughlin C. The correlation of early hyperglycemia with outcomes in adult trauma patients: a systematic review. J Spec Oper Med. 2013 Winter;13(4):34-9.
http://www.ncbi.nlm.nih.gov/pubmed/24227559?tool=bestpractice.com
[67]Penney DG, Helfman CC, Dunbar JC Jr, et al. Acute severe carbon monoxide exposure in the rat: effects of hyperglycemia and hypoglycemia on mortality, recovery, and neurologic deficit. Can J Physiol Pharmacol. 1991 Aug;69(8):1168-77.
http://www.ncbi.nlm.nih.gov/pubmed/1782598?tool=bestpractice.com
Treating hyperglycemia with insulin is therefore appropriate. It is critical, however, to avoid dangerously low levels of blood glucose through over-aggressive treatment of hyperglycemia.[68]Garcia-Ballestas E, Villafañe J, Nuñez-Baez K, et al. A systematic review and meta-analysis on glycemic control in traumatic brain injury. Clin Neurol Neurosurg. 2024 Oct;245:108504.
https://www.doi.org/10.1016/j.clineuro.2024.108504
http://www.ncbi.nlm.nih.gov/pubmed/39141934?tool=bestpractice.com