History and exam

Key diagnostic factors

common

exposure to incomplete combustion of carbon-containing material

A variety of sources generate CO, including internal combustion engines, water heaters, and home heating units. Kerosene space heaters were responsible for CO poisoning in the 1970s. Other sources of CO exposure are the paper mill industry, steel mills, explosives used in construction, petroleum refineries, and gasoline-or propane-powered machinery.[12][22][23]

exposure to fire with smoke inhalation

Up to 75% of fire-related injuries are associated with CO poisoning.[5][4] Concomitant cyanide poisoning should be considered in these cases of CO poisoning.[2][24]

nausea

Nonspecific symptoms may occur.

headache

Nonspecific symptoms may occur.

vomiting

Nonspecific symptoms may occur.

blurred vision

Nonspecific symptoms may occur.

dizziness

Nonspecific symptoms may occur.

vertigo

Nonspecific symptoms may occur.

altered consciousness

Nonspecific symptoms, including drowsiness, tiredness, syncope, or confusion, may occur. Lethargy/mild tiredness can be a feature of chronic poisoning.[1]​ Coma may be present in people with severe poisoning.

dyspnea

Breathing difficulties or shortness of breath may occur.[1]

tachycardia

Nonspecific cardiovascular signs are a common feature.

hypotension or hypertension

Nonspecific cardiovascular signs are a common feature.

cardiac arrhythmias

Nonspecific cardiovascular signs are a common feature.

pain

Chest or abdominal pain may occur.

weakness

In one epidemiologic study, subjective weakness was reported in around 20% of patients with acute CO poisoning.[39]

sleep changes

Patient may be experiencing tiredness, fatigue, and sleep disturbance.[1]

irritability

May indicate severe poisoning.[27]

flu-like symptoms

A feature of chronic CO poisoning.[1]

Other diagnostic factors

common

exposure to methylene chloride

CO is formed during metabolism of methylene dichloride (dichloromethane). Exposure to CO during paint or furniture stripping and degreasing may result in poisoning.[25]

delayed neuropsychiatric features

May be present in a patient previously exposed to CO. Features include memory impairment, disorientation, apathy, mutism, irritability, inability to concentrate, emotional lability, peripheral neuropathy, incontinence, choreoathetosis, apraxia, psychosis, dementia, Parkinsonism, and hearing loss.[27][40][41]

other severe neurologic symptoms

Seizures, coma, cortical blindness, or cerebral edema may develop in patients with severe poisoning.[1][27][40]

cutaneous blistering

Nonspecific symptoms may occur.

pulmonary edema

Symptoms include shortness of breath.

uncommon

cardiac arrest

A feature of severe CO poisoning.

focal neurologic abnormalities (e.g., hemiplegia, truncal ataxia)

Neurologic symptoms include acute stroke-like symptoms. Truncal ataxia is a common physical sign in those who are conscious.

disseminated intravascular coagulation

May be a feature of CO poisoning.[42]​ See Disseminated intravascular coagulation.

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