History and exam
Key diagnostic factors
common
haemoglobinuria
Budd-Chiari syndrome
Other diagnostic factors
common
history of aplastic anaemia
fatigue
Almost all patients with PNH, regardless of the degree of anaemia, have fatigue and listlessness.[1]
anaemia
More than 90% of patients with PNH are anaemic at presentation. Degree of anaemia varies greatly and is accompanied by signs of intravascular haemolysis (increased reticulocyte count, increased serum LDH, low haptoglobin).[18] In descending order, haemolysis, iron deficiency, and concomitant bone marrow hypoplasia contribute to the anaemia of PNH patients.
abdominal pain
dysphagia and odynophagia
Difficult or painful swallowing due to increased oesophageal contractions. Symptoms worsen during haemolytic episodes.
erectile dysfunction
Almost all men have erectile dysfunction, especially during paroxysms of haemoglobinuria.[9]
dyspnoea
Many patients have dyspnoea, which may be due to anaemia or pulmonary hypertension.[9]
infections
Moderately common due to granulocytopoenia.[15]
uncommon
bleeding diathesis
Petechiae, ecchymoses, or frank bleeding (epistaxis, mucosal bleeding, gastrointestinal bleeding, genitourinary bleeding) may occur in the setting of concomitant marrow hypoplasia or myelodysplastic syndrome with low platelet numbers.
neurological signs and symptoms
Usually related to cerebrovascular thrombotic events. Signs of elevated intracranial pressure (headache/vomiting, papilloedema, coma in severe cases) and/or frank sensorimotor deficits according to the affected site.[20]
Risk factors
strong
aplastic anaemia/hypoplastic bone marrow
About 50% of patients with aplastic anaemia have a clone of paroxysmal nocturnal haemoglobinuria (PNH) cells, predominantly small and asymptomatic.[7] Expansion of the clone, sufficient to cause symptoms, occurs in about 15% of patients with aplastic anaemia successfully treated without stem cell transplantations.[12] The reason for the expansion of the clone(s) in these patients is unknown.
The presence of a PNH clone, regardless of its size, has been associated with a favourable response to immunosuppressive therapy (IST) and stem cell transplantation (SCT) in aplastic anaemia. Patients with PNH positivity showed higher success rates for IST response and haematopoietic SCT outcomes compared with those without PNH. Additionally, PNH positivity had a beneficial impact on clonal evolution.[13]
All patients with PNH have a degree of marrow hypoplasia, often detected only by marrow culture studies.[14] Patients with classic PNH may have bone marrow failure as a late event.[15]
weak
myelodysplasia
Small PNH clones have been described in patients with low-risk myelodysplasia, particularly refractory anaemia with a hypoplastic marrow. Clinical PNH arises in a few of these patients.[16][17] The presence of a PNH clone, regardless of its size, has been associated with a favourable response to IST and stem cell transplantation in myelodysplastic syndromes.[13]
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