Investigations

1st investigations to order

pulse oximetry

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Helpful for initial assessment.

Pulse oximetry under-detects hypoxaemia in the setting of carboxyhaemoglobin, and will appear normal even in patients with significantly elevated carboxyhaemoglobin (CO-Hb) levels.

Result

hypoxaemia

arterial blood gas

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Result
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Severe acidosis may suggest hypoperfusion from shock, carbon monoxide poisoning, or cyanide toxicity.

Result

may show severe metabolic acidosis

carboxyhaemoglobin (CO-Hb) level

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Normal levels are around 1% to 3% and in smokers up to 10%. Carbon monoxide (CO)-oximetry should be used to measure CO-Hb levels.

Arterial sampling is not necessary as there is a high correlation between venous and arterial samples.

Result

CO-Hb level >15% in carbon monoxide poisoning

chest x-ray

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Air trapping and atelectasis suggest airway injury, obstruction, and collapse.

Pulmonary oedema may suggest acute respiratory distress syndrome or cardiogenic oedema.

Result

air trapping; atelectasis; airspace opacity (oedema)

ECG

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Increased cardiac demand, inflammation, and systemic and cellular hypoxia associated with burn and inhalation injury all predispose to cardiac ischaemia.

Result

arrhythmias, ischaemia

cardiac telemetry monitoring

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Increased cardiac demand, inflammation, and systemic and cellular hypoxia all predispose to cardiac ischaemia or arrhythmias.

Result

arrhythmias

urine toxicology screen

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There is a high incidence of intoxication among victims of residential fires in particular.[24]

Result

evidence of illicit drug use

serum ethanol level

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Result
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There is a high incidence of intoxication among victims of residential fires in particular.[24]

Result

elevated blood alcohol level in intoxicated patients

Investigations to consider

pulmonary function tests (PFT)

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If available, PFT may allow characterisation and serial measurement of airflow obstruction.

Result

Decreased FEV1 or forced vital capacity; flow-volume loop suggesting obstruction

laryngoscopy and bronchoscopy

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Many practitioners advocate these procedures to assess airway oedema, although clear evidence of benefit is lacking.[19]

Result

Airway oedema, burn, or ulceration; debris in airways

serum lactate

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Result
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While often interpreted as evidence of cyanide toxicity, this test is non-specific and probably represents hypoperfusion and carbon monoxide poisoning more commonly.[20]

Result

Elevation of lactate level

Emerging tests

cyanide level

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Result
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A rapid point-of-care test is not commercially available, but a simple optical analyser has been developed which accurately detects cyanide levels of imminent concern in around 2 minutes, as well as a paper microfluidic device that successfully measured cyanide ions in the blood samples of 20 fire survivors.[20][22][23]

Result

level greater than 11.62 micromol/L (0.5 mg/L)

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