History and exam

Key diagnostic factors

common

presence of risk factors

Risk factors include cirrhosis, acute hepatic failure and portacaval shunt.

mood disturbances

Patients may experience euphoria or depression.

sleep disturbances

Patients may have sleep disturbances such as insomnia or hypersomnia.

motor disturbances

Patients may have motor disturbances such as ataxia, and extrapyramidal symptoms such as muscle rigidity, bradykinesia, hypokinesia, slow monotonous speech, and parkinsonian-like tremor.[1]

advanced neurological deficits

Patients may have somnolence and confusion, which may be followed by unconsciousness or coma. Hyper-reflexia, nystagmus, and clonus may be present in rare cases.

Other diagnostic factors

common

asterixis

Negative myoclonus, detected by extending the arms, dorsiflexing the wrist, and spreading the fingers. Not specific for HE.

palmar erythema

Physical examination findings consistent with chronic liver disease may also be present.

spider angiomata

Physical examination findings consistent with chronic liver disease may also be present.

peripheral oedema

Physical examination findings consistent with chronic liver disease may also be present.

jaundice

Physical examination findings consistent with chronic liver disease may also be present.

hepatomegaly

An enlarged liver may be seen in early viral hepatitis.

ascites

Other physical examination findings consistent with chronic liver disease may also be present.

Risk factors

strong

hypovolaemia

May precipitate episodic HE.[16][17]

gastrointestinal bleeding

May precipitate episodic HE.[16][17]

constipation

May precipitate episodic HE.[16] The mechanism is thought to be increased absorption of nitrogenous compounds from the gut.

excessive protein intake

May precipitate episodic HE.[16] The mechanism is thought to be increased absorption of nitrogenous compounds from the gut.

hypokalaemia

May precipitate episodic HE.[16][17]

hyponatraemia

Independent risk factor for HE in patients with cirrhosis.[1][18]

metabolic alkalosis

May precipitate episodic HE.[16][17]

hypoxia

May precipitate episodic HE.[16][17]

sedative use

May precipitate episodic HE.[16][17]

opioid use

May precipitate episodic HE.[19]

diuretic overdose

May precipitate episodic HE.[19]

hypoglycaemia

May precipitate episodic HE.[16][17]

infection

May precipitate episodic HE.[16][17]

hepatic or portal vein thrombosis

May precipitate episodic HE.[16][17]

portacaval shunt

The incidence of HE in the 2 years after transjugular intrahepatic portosystemic shunt (TIPS) placement is up to 55%.[4]

Older patients (>65 years), those with more advanced liver disease, and those with prior episodes of HE may be at higher risk for developing HE after TIPS.

renal impairment

Patients with cirrhosis and renal impairment have a higher risk of HE.[20]

Use of this content is subject to our disclaimer