Differentials

hypoglycemia due to nondiabetic drugs

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Drug-induced hypoglycemia in people without diabetes is more common in those who are older and have underlying comorbidities.[41]​ Nondiabetic drugs that may cause hypoglycemia include:[41]

  • Certain antibiotics, particularly sulfonamides and fluoroquinolones.

  • Beta-blockers. These can mask symptoms and signs of hypoglycemia, which can predispose patients to severe, life- threatening hypoglycemia.[61]​ Nonselective beta-blockers can also impair hepatic and renal release of glucose into the circulation, which can cause hypoglycemia.[41]

  • Quinine.

  • Pentamidine.

  • Indomethacin.

This list of drugs is not exhaustive, and you should consult a drug formulary for more information.

Alcohol can cause hypoglycemia during and after consumption, particularly if the patient has not eaten recently.[3][4][17][37]​ In addition, alcohol-related liver disease can lead to hepatic insufficiency, which decreases gluconeogenesis.

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Alcohol-related hypoglycemia is usually associated with elevated levels of beta-hydroxybutyrate, and low insulin and C-peptide levels.[41]

acute or chronic liver failure

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The patient may be asymptomatic, or have features of underlying liver disease, such as right upper quadrant pain, weight loss, pruritus, fatigue, or anorexia. Specifically, patients with acute liver failure will have jaundice and encephalopathy. The patient may also have a history of liver disease, or risk factors for liver disease.

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Elevated liver function tests (AST and ALT), coagulopathy.

chronic kidney disease

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The patient may have vague symptoms such as fatigue and edema.

The patient may have a history of chronic kidney disease, and risk factors for renal impairment.

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Elevated BUN and creatinine levels.

celiac disease

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There are usually unexplained gastrointestinal symptoms (e.g., chronic diarrhea), weight loss, features of malabsorption, or a skin rash consistent with dermatitis herpetiformis.[83][84][85]​​​​​​​

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Elevated immunoglobulin A-tissue transglutaminase (IgA-tTG) and normal or low quantitative IgA titers.[85][86]​​

If dermatitis herpetiformis is present, skin biopsy will show granular deposits of IgA at the dermal papillae of lesional and perilesional skin by direct immunofluorescence.

sepsis

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Acute deterioration in a patient in whom there is clinical evidence or strong suspicion of infection.[87]​ Take into account that people with sepsis may have nonspecific, nonlocalized presentations, such as feeling very unwell, and may not have a high temperature.[87]

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Blood or other body fluid cultures may be positive for the infectious organism.

White cell count elevated.

inanition

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The patient may have a history of eating disorders such as anorexia nervosa, mental health conditions, diseases causing malabsorption, alcohol and substance use disorders, or a period of starvation.[41]

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Clinical diagnosis.

primary adrenal insufficiency

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Features of primary adrenal insufficiency (Addison disease) include progressive substantial fatigue and generalized weakness associated with mucocutaneous hyperpigmentation, hypotension and/or postural hypotension, and salt craving.

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Low morning cortisol level.[88]

Elevated plasma adrenocorticotropic hormone level.[89][90]​​​

hypothyroidism

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Features include lethargy, fatigue, change in voice, cold intolerance, constipation, weight gain, skin changes, hair loss, and bradycardia.[91]​ If central hypothyroidism is present, there may also be features of hypopituitarism, including hypogonadism and secondary adrenal insufficiency.

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Inappropriately low, normal, or elevated thyroid-stimulating hormone, depending on the type of hypothyroidism.

Low free thyroxine (T4).

hypopituitarism

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The clinical manifestations of hypopituitarism are variable and dependent on the duration and degree of hormone deficiencies, as well as the age of onset. Key risk factors for hypopituitarism include pituitary tumors, apoplexy, surgery, or radiation; genetic disorders such as mutations of the PROP1 gene that cause familial hormone deficiencies; and hypothalamic disease.

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Results of investigations will depend on the underlying cause of hypopituitarism.

non-islet cell tumors (NICT)

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Also known as insulin-like growth factor-2 (IGF-2)-secreting tumors.[92][93]​​​

There may be features of the underlying tumor. Tumors associated with NICT hypoglycemia are hepatocellular carcinoma, fibrosarcoma, mesothelioma, adrenocortical carcinoma, stomach carcinoma, pancreatic carcinoma, medullary thyroid carcinoma, lymphoma, leukemia, carcinoid syndrome.[94]

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Blood tests will show:[95]

  • Insulin level <3 microunits/mL

  • Proinsulin level <5 picomol/L

  • C-peptide level <0.2 nanomol/L

  • Insulin-like growth factor 1 (IGF-1) level <100 nanograms/mL

  • Insulin-like growth factor 2 (IGF-2) level >275 nanograms/mL

  • IGF-2:IGF-1 ratio >3:1.

Further investigation specific to the underlying tumor will be required.

insulinoma

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An insulinoma is an insulin-secreting tumor.[92][93]

It is difficult to clinically distinguish hypoglycemia due to insulinoma from diabetic hypoglycemia because the patient typically presents in a similar manner, with typical neuroglycopenic symptoms that improve quickly with administration of glucose.[92][93]

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48-hour supervised fast may demonstrate:

  • Whipple's triad (biochemical hypoglycemia, symptoms consistent with hypoglycemia, and reversal with carbohydrate replacement), and

  • Inadequately suppressed insulin levels (>3 micro-IU/mL) or, in some instances, inadequately suppressed proinsulin levels (≥5 picomol/L).[96]

There has been some debate as to whether a 48-hour fast under observation is adequate in rendering a diagnosis, and the full 72-hour supervised fast has been found to identify individuals with an insulinoma not identified by 48-hour fast.[97]

72-hour hour fast may show:[98]

  • Insulin >5 milli-IU/L (>36 picomol/L)

  • C-peptide >0.6 nanograms/mL (>0.2 nanomol/L)

  • Insulin:C-peptide ratio <1.0

  • Proinsulin >20 picomol/L

  • Absence of sulfonylurea metabolites in plasma or urine

CT or MRI will determine exact location of an insulinoma, its relationship to vital structures, and any metastases.[98]

non-insulinoma pancreatogenous hypoglycemia (NIPHS)

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Clinical history of postprandial hypoglycemia, or hypoglycemia following bypass surgery.[99][100]​​​

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Normal pancreatic imaging studies.[99]

Positive selective arterial calcium stimulation test.

Histopathologic examination of the pancreas will show nesidioblastosis (islet hypertrophy and hyperplasia, and enlarged and hyperchromatic beta-cell nuclei).[99][100]

post total pancreatectomy with islet auto-transplantation (TP-IAT)

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Clinical history of hypoglycemia occuring after TP-IAT.[101]

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Clinical diagnosis.

post gastric bypass surgery

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Gastrointestinal symptoms (e.g., nausea, vomiting, diarrhea, belching) or vasomotor symptoms (e.g., shock, syncope, near-syncope, palpitations, dizziness, desire to lie down, diaphoresis).[99]

Patients may have had bariatric surgery for treatment of obesity; Roux-en-Y gastric bypass surgery is most common.[99]

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Low blood glucose after a carbohydrate load (oral glucose tolerance) or meal.

dumping syndrome

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Gastrointestinal symptoms (e.g., nausea, vomiting, diarrhea, belching) or vasomotor symptoms (e.g., shock, syncope, near-syncope, palpitations, dizziness, desire to lie down, diaphoresis).[99]

Dumping syndrome occurs in patients who have had gastric surgery; hypoglycemia is related to abnormal transport of food to the small intestine. Use the Dumping Symptom Rating Scale, which is a self-assessment questionnaire, to assess the likelihood of dumping syndrome.[99] 

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Low blood glucose after a carbohydrate load (oral glucose tolerance) or meal.

rapid gastric emptying

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Gastrointestinal symptoms (e.g., nausea, vomiting, diarrhea, belching) or vasomotor symptoms (e.g., shock, syncope, near-syncope, palpitations, dizziness, desire to lie down, diaphoresis).[99]

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Low blood glucose after a carbohydrate load (oral glucose tolerance) or meal.

insulin autoimmune syndrome

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Extremely rare condition, apart from in Japan and other Asian countries.[102]​ Usually spontaneously resolves.[102] There may be a past medical history of other autoimmune diseases.[102]

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Elevated insulin antibody levels in the absence of exposure to exogenous insulin; elevated insulin receptor antibody levels.[102]

accidental, surreptitious, or malicious hypoglycemia, including Munchausen syndrome by proxy

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Accidental, surreptitious, or malicious hypoglycemia due to administration of insulin or insulin secretagogs should be considered as part of history and examination.[103]

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Diagnosis of exclusion.

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