History and exam

Key diagnostic factors

common

age over 50 years

The prevalence of stress incontinence peaks in the fifth decade of life, whereas the prevalence of both mixed and urgency incontinence continues to increase with age.​[11]

BMI over 25

Excess weight, especially in women with a BMI over 25, increases pressure on pelvic tissues, causing chronic strain, stretching, and weakening of the muscles, nerves, and other pelvic structures.[3]

increased parity

Increasing parity, vaginal delivery, and episiotomy are associated with stress incontinence.[3][14]

involuntary urine leakage on effort, exertion, sneezing, or coughing

Suggestive of stress incontinence.

involuntary urine leakage accompanied by or immediately preceded by urgency

Suggestive of urgency incontinence.

frequency of urination

Occurs in overactive bladder.

nocturia

Occurs in overactive bladder.

use of drugs that can cause urinary incontinence

​Includes drugs with anticholinergic effects, such as antihistamines, antidepressants, and antipsychotics.[11]​​ Calcium-channel blockers and alpha-blockers have also been linked to urinary retention and difficulty in voiding. Diuretics may cause polyuria, frequency, and urgency.[11]​​​ Consult your local drug information source for more information on drugs that can cause urinary incontinence.

Other diagnostic factors

common

suggestive bladder diary

A 3-day diary records each void throughout the day and fluid intake. Documentation of incontinence events, times at which they occur, and any associated symptoms or events preceding leakage (i.e., urgency or sneezing).

vaginal bulge/pressure

Pelvic organ prolapse indicates lack of tissue support to vagina and neighboring organs, such as the bladder. An enlarged uterus may cause abnormal voiding or stress incontinence.

urogenital atrophy

Menopause and aging are strongly associated with the emergence or worsening of lower urinary tract dysfunction.[35]​ Thin, pale, poorly vascularized urogenital tissue indicates a deficiency in circulating estrogen to the urogenital tissues.[35][36]

uterine prolapse or posterior support weakness

Other forms of prolapse (e.g., uterine prolapse or posterior support weakness such as rectocele) can affect bladder function. They may cause bladder outlet obstruction and resultant incontinence or retention.

long-term residence in a care facility

High correlation with chronic medical comorbidities (e.g., bowel disease, chronic cough [e.g., COPD, asthma], arthritis, diabetes mellitus, and cardiovascular disease [e.g., hypertension, heart failure]) that may be associated with incontinence has been observed.[19]​ For individuals in a care facility, constipation, cardiovascular disease (especially heart failure), immobility, and wheelchair dependence increases the rate of urinary incontinence.[39]​ Cognitive impairment and dementia is highly prevalent in older people in care homes with urinary incontinence.[40]

uncommon

chronic heart failure

Chronic medical conditions may be associated with incontinence.​[11][19]

diabetes mellitus

Chronic medical conditions may be associated with incontinence.[11]​​[19]​ Diabetes commonly results in overactive bladder and can lead to sensory neurogenic bladder, which predisposes to incontinence.

excess fluid intake

May be associated with increased urine output.

cognitive impairment on mental state exam

Abnormalities (e.g., memory deficits) may be suggestive of dementia, which is associated with an increased incidence of urinary incontinence in older women.

history of neurologic disease

Neurologic diseases, such as cerebrovascular accidents, multiple sclerosis, and Parkinson disease, may affect the neuronal pathways of the genitourinary system, causing urinary urgency or incontinence, or impair mobility, resulting in functional incontinence.[27]

history of back injury

Suggests possible spinal cord injury as a cause of urinary incontinence.[11]​​

history of recurrent urinary tract infections

May suggest an inflammatory process of the lower urinary tract caused by bacteria that also causes urinary incontinence.[11]​​

dysuria

Suggestive of a urinary tract infection as a cause of urinary incontinence.​[11]

hematuria

May suggest an alternative lower urinary tract pathology which contributes to urinary urgency, frequency, and urgency incontinence.[11]​​ Importantly, the presence of hematuria without identifiable etiology prompts workup for bladder cancer, especially in women with increased risk factors of age >65 years, tobacco use, occupational exposure, or persistent irritative urinary symptoms (urgency, frequency, dysuria, and incontinence).

post-void dribbling

May suggest an alternative lower urinary tract pathology, such as urethral diverticula, as a cause of urinary incontinence.​[11]

pooling of urine in vaginal tract

Suggests a genitourinary fistula as a cause of urinary incontinence.​[11]

urethral discharge or tenderness

May suggest urethral diverticulum, carcinoma, or inflammation, which also causes urinary incontinence.[11]​​

enlarged uterus

A bimanual examination also provides valuable information about the size and conformation of the pelvic organs, if present. Mechanical compression of the bladder by an enlarged, bulky uterus may cause urinary urgency and frequency by constricting the bladder's ability to distend in the already occupied pelvis. Causes mechanical compression of the bladder causing urinary urgency and frequency.

loss of perineal sensation

Suggests disruption of neurologic input to the perineum.

abnormal bulbocavernosus and wink reflexes

Anal reflex is performed by gently stroking the skin lateral to the anus. In normal circumstances, an anal wink, or contraction of the anus, is visualized. Similarly, the clitoro-anal reflex, which involves tapping the clitoris, causes contraction of the bulbocavernosus, ischiocavernosus, and anal sphincter. Abnormal reflexes suggest disruption of the sacral reflex.

weakened sphincter tone

Reflects poor pelvic floor muscle support to urethral sphincter.

fecal impaction

Sign of pelvic floor dysfunction, manifested by anorectal changes.

Risk factors

strong

increasing age

Incidence and prevalence increase with age.[7][8]​​​​​​[11]​ The prevalence of stress incontinence peaks in the fifth decade of life, whereas the prevalence of both mixed and urgency incontinence continues to increase with age.​​[11]

pregnancy

Increasing parity, vaginal delivery, and episiotomy are associated with stress incontinence.[3][14][15][16]​ This is due to weakening and stretching of muscles and connective tissue during delivery, as well as damage to pudendal and pelvic nerves.[16]

obesity

Excess weight, especially in women with a BMI over 25, increases pressure on pelvic tissues, causing chronic strain, stretching, and weakening of the muscles, nerves, and other pelvic structures.​[3]​​[33]

pelvic organ prolapse

Commonly coexistent condition with urinary incontinence and pelvic floor dysfunction.[34]

postmenopausal status

Menopause and aging are strongly associated with the emergence or worsening of lower urinary tract dysfunction.[35]​ Estrogen depletion results in atrophy of the superficial and intermediate layers of the urethral mucosal epithelium.[36]​ This causes atrophic urethritis, diminished urethral mucosal seal, loss of compliance, and irritation, possibly leading to either stress or urgency incontinence.

diuretic use

May cause polyuria, frequency, and urgency.​[11]

caffeine consumption

May cause frequency and urgency.​[37]

constipation

Women with urinary incontinence are significantly more likely to report bowel symptoms (constipation, fecal incontinence).[17]​ Constipation with chronic repeated, prolonged defecatory straining efforts contribute to progressive neuropathy and dysfunction.[18]​ Concomitant urinary incontinence and chronic constipation in young women with longstanding history of urinary incontinence may indicate occult spina bifida.

fecal incontinence

Commonly coexistent condition with urinary incontinence, particularly in older people and in nursing home residents.[17][19][20]

high-impact physical activity

Strenuous activity increases stress on pelvic support structures, which leads to stretching and weakening of the muscles, nerves, and other pelvic structures and results in urinary incontinence.[21][22]

obstructive sleep apnea

​Elevated levels of atrial natriuretic peptide are produced during apneic episodes, with increased abdominal pressure leading to increased urine production, and may cause nocturia and nocturnal polyuria.[11]​​[38]

long-term residence in a care facility

High correlation with chronic medical comorbidities (e.g., bowel disease, chronic cough [e.g., COPD, asthma], arthritis, diabetes mellitus, and cardiovascular disease [e.g., hypertension, heart failure]) that may be associated with incontinence has been observed.[19]​ For individuals in a care facility, constipation, cardiovascular disease (especially heart failure), immobility, and wheelchair dependence increases the rate of urinary incontinence.[39]​ Cognitive impairment and dementia is highly prevalent in older people in care homes with urinary incontinence.[40]

dementia

Associated with increased incidence of urinary incontinence in older women. Types of dementia may include Lewy body disease, vascular dementia, normal pressure hydrocephalus, frontotemporal dementia, and Alzheimer disease.​[27]​ Incontinence may be due to neurologic problems, behavioral problems, or myogenic changes, but also from the cognitive impairment and impaired mobility often seen in dementia.[27]

stroke and other central nervous system/spinal disorders

Interruption of central nervous system inhibitory pathways is associated with lower urinary tract dysfunction.[26]​ Detrusor hyperreflexia resulting from upper motor neuron lesions after stroke may present as urinary urgency and urgency incontinence.[27][29]

Parkinson disease

Urinary incontinence may be a direct consequence of neurologic damage caused by Parkinson disease or may be caused indirectly as a result of physical limitations imposed by the disease.[27][29]​​ Urinary dysfunction in Parkinson disease is predominantly associated with detrusor overactivity (urgency, frequency, nocturia, and urge incontinence).[41]​ External sphincter bradykinesia, which may contribute to urinary obstruction, is rare.[42]

multiple sclerosis

Up to 90% of patients with multiple sclerosis experience neurogenic lower urinary tract symptoms (most commonly detrusor overactivity) over the course of their disease.[43]

weak

white women

A study on different racial/ethnic groups within the US determined that stress and mixed urinary incontinence were significantly less prevalent in black versus white women; however, there was no significant difference in prevalence of urgency urinary incontinence between the groups.[6]

functional impairment

Mobility impairment or sensory impairments (i.e., impaired vision) contributing to mobility limitation are associated with incontinence.[11]​​[27]​ The relationship between functional impairment and condition is unclear. Possibilities include difficulties in getting to the bathroom and removing clothing, or the condition may be a consequence of general frailty in older patients or an underlying systemic illness, such as cerebrovascular accident.

family history of incontinence

Increased risk of urinary incontinence and severe symptoms if first-degree relatives are affected.[44][45][46]

childhood enuresis

Correlation with adult urinary incontinence observed.[47][48]

chronic cough

In chronic coughing, the rapid increase in intra-abdominal pressure and impact loading on pelvic musculature and connective tissue can create damage over time.[19]

diabetes mellitus

The pathogenesis of diabetes-induced bladder dysfunction is multifactorial.[49]​ Diabetes commonly results in overactive bladder and can lead to sensory neurogenic bladder, which predisposes to incontinence.

depression

The exact nature of the relationship between bladder symptoms and depression remains unknown, but may be due to altered serotonin function.[50][51]

chronic heart failure

The association between heart failure and urinary symptoms may be directly attributable to worsening heart failure pathophysiology; however, management strategies for heart failure, in particular diuretics, ACE inhibitors, and beta-blockers, may contribute to the risk of developing urinary incontinence.[52]​ There is also significant overlap in the known risk factors associated with urinary incontinence and heart failure, such as increasing age, and the comorbid illnesses of diabetes, obstructive sleep apnea, obesity, and depression.[53]

smoking

May contribute to chronic coughing and interfere with collagen synthesis.​[54]

genitourinary and pelvic surgery

Musculofascial attachments of the bladder to the surrounding pelvic wall can be disrupted during procedure. Nerve damage may also occur during some procedures (e.g., hysterectomy).[55] Additionally, abdominopelvic surgery is associated with an inherent risk of direct urinary tract injury, which may lead to urinary fistula.

radiation exposure

May cause injury to connective tissue, resulting in decreased elasticity and mobility (lead pipe urethra), which may lead to stress incontinence. Decreased bladder compliance from radiation may lead to low bladder capacity and mucosal irritation, resulting in irritative bladder symptoms and urinary urgency.[56] Urinary fistulae may also form after pelvic irradiation.

alcohol consumption

The combination of impaired mobility, confusion associated with alcohol use, and sedation, in conjunction with increased diuresis, can contribute to urinary incontinence.

antihistamine use

Those with anticholinergic effects have been associated with dry mouth, which, in turn, leads to increased fluid intake and the potential for urinary retention through inhibition of the contractile activity of the bladder, leading to incontinence. May also cause sedation.​[11]

sedative use

Causes sedation, muscle relaxation, and confusion.​[11]

hypnotic use

Causes sedation, muscle relaxation, and confusion.[11]​​

opioid analgesic use

Has been associated with urinary retention, fecal impaction, sedation, and delirium. May lead to incontinence.[11]​​

anticholinergic use

Has been associated with dry mouth, which, in turn, leads to increased fluid intake and the potential for urinary retention through inhibition of the contractile activity of the bladder, leading to incontinence. Also causes sedation.​[11]

antidepressant use

The anticholinergic action of some antidepressants causes suppression of bladder contraction and alpha-adrenergic-mediated increased bladder neck tone. Resultant increased bladder outlet resistance is manifested by urinary retention, urinary urgency, frequency, and incontinence.[11]​​

Selective serotonin-reuptake inhibitors (SSRIs) increase cholinergic release resulting in urgency and urgency incontinence.[11]​​

antipsychotic use

Anticholinergic action of some antipsychotics causes suppression of bladder contraction and alpha-receptor-mediated increased bladder neck tone. Resultant increased bladder outlet resistance is manifested by urinary retention, urinary urgency, frequency, and incontinence. Also causes sedation.[11]​​

alpha-blocker use

Has been associated with urinary retention and voiding difficulty, which can lead to incontinence.[11]​​​

calcium-channel blocker use

Has been associated with urinary retention and voiding difficulty, which can lead to incontinence.[11]​​

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