Investigations

1st investigations to order

full blood count with differential

Test
Result
Test

Pancytopenia is common, but the diagnosis of AA requires only two cytopenias among white cell, red cell, and platelet lineages.

Presence of macrocytosis may suggest inherited syndrome (Fanconi anaemia or dyskeratosis congenita).

Presence of monocytopenia may indicate inherited GATA2-related disorder.

Result

≥2 cytopenias among the following: Hb <100 g/L (<10 g/dL), platelet <50 × 10⁹/L, and/or absolute neutrophil count <1.5 × 10⁹/L

reticulocyte count

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Result
Test

A low reticulocyte count identifies anaemia as being hypoproductive, as opposed to hyperproductive anaemias such as haemolytic anaemia.

Result

corrected reticulocyte percentage <1% or absolute reticulocyte count <20 × 10⁹/L (<60 × 10⁹/L using an automated analysis)

bone marrow biopsy and cytogenetic analyses

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Result
Test

A hypocellular marrow is a definitive diagnostic finding for AA. Moreover, there should be an absence of abnormal cell population (such as blasts) and no fibrosis.

Result

hypocellular marrow with no abnormal cell population

Investigations to consider

serum B12 and folate levels

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Result
Test

Severe vitamin B12 and/or folate deficiency may be an alternative cause of pancytopenia but should not preclude or delay bone marrow analysis.

Result

normal

HIV testing

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Result
Test

May be an alternative cause of pancytopenia, but should not preclude or delay bone marrow analysis.

Result

negative

liver function tests (LFTs)

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Result
Test

Abnormal LFTs may suggest an inherited syndrome (dyskeratosis congenita or Shwachman-Diamond syndrome), recent hepatitis, or exposure to toxins.[5][6] Elevated lactate dehydrogenase suggests haemolysis.

Result

usually normal

autoantibody screen

Test
Result
Test

May indicate an associated autoimmune disorder. Anecdotal reports exist for an association between AA and systemic lupus erythematosus, thymoma, eosinophilic fasciitis, and coeliac disease.[29] The mechanism underlying this association is unclear.

Result

usually normal

flow cytometry for glycosylphosphatidylinositol (GPI)-anchored proteins

Test
Result
Test

Small paroxysmal nocturnal haemoglobinuria (PNH) clones may be detected in red blood cells, monocytes, and granulocytes in up to 50% of patients.[32]

Result

variable

chest x-ray

Test
Result
Test

Ordered if malignancy, infection, or lung fibrosis is suspected.

Result

normal, unless infection is present

abdominal ultrasound

Test
Result
Test

Ordered if malignancy, Fanconi anaemia, or telomeropathy is suspected.

Abnormal or displaced kidneys may be seen in Fanconi anaemia.

Splenomegaly or lymphadenopathy raises possibility of a haematological malignancy rather than AA.

Liver cirrhosis/fibrosis suggests telomeropathies.

Result

variable

appropriate genetic tests

Test
Result
Test

If a congenital syndrome is suspected, appropriate diagnostic tests should be obtained: for example, chromosomal breakage testing (diepoxybutane [DEB] test) for Fanconi anaemia; relevant genetic sequencing for other disorders, such as TERC, TERT, TINF2, DKC1 (for dyskeratosis congenita), SBDS (for Shwachman-Diamond syndrome), and GATA2 (for inherited GATA2-related disorder). Telomeropathies are screened for by measuring telomere length.

Telomere length may be measured to rule out telomeropathies, although this test is not routinely available in many centres.

Inherited marrow failure syndrome gene panels with next-generation sequencing are available at some specialist centres. They can be used to identify telomeropathies, mutations, and malignancies.

Result

variable: positive result for Fanconi anaemia; DKC1 mutation indicates X-linked dyskeratosis congenita; TERC, TERT, and TINF mutations are seen in autosomal-dominant dyskeratosis congenita; heterozygous germline GATA2 mutations are seen in inherited GATA2-related disorders; telomere length below first percentile indicates telomeropathies

computed tomography scan

Test
Result
Test

Ordered if telomeropathies or dyskeratosis congenita is suspected.

Lung fibrosis, cirrhosis, or non-cirrhotic portal hypertension may suggest dyskeratosis congenita.

Result

variable

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