History and exam

Key diagnostic factors

common

presence of risk factors

The key risk factors are soil exposure in or migrants from an endemic area of the world.[11][12][13][14][33][16]

Risk of life-threatening hyperinfection is primarily related to immunosuppression, particularly with corticosteroids.[20][21]

infection with other parasites

Co-infection with other parasites is common.[11]

Strongyloides infection is suggested by persisting eosinophilia after treatment for other parasites.

Giardia with eosinophilia necessitates that the diagnosis of strongyloides infection is considered.

Other diagnostic factors

common

abdominal pain

Reported by 40% of patients with chronic infection.[11]

May be misdiagnosed as irritable bowel syndrome or somatic symptom disorder.

altered bowel habit

20% of patients report stool changes such as diarrhoea or constipation.[11]

weight loss

Reported by 18% of patients.[11]

fever (hyperinfection)

A sign of hyperinfection.

signs of sepsis (hyperinfection)

In hyperinfection, patients often rapidly become critically ill with bacteraemia and sepsis caused by an enteric organism, such as Escherichia coli or enterococcus, and develop signs of septic shock.

uncommon

chronic cough

Found in 14% of patients.[11]

May be misdiagnosed as asthma.

wheezing

Found in 10% of patients.[11]

An immigrant presenting with new-onset wheezing and eosinophilia should be evaluated or treated for strongyloides infection before any corticosteroids are given.

Corticosteroids given as empirical asthma therapy can precipitate life-threatening hyperinfection.

pruritus or dermatitis

Found in 14% of patients.[11]May be misdiagnosed with psychogenic pruritus.

[Figure caption and citation for the preceding image starts]: A creeping dermatological eruption on the back of a patient with strongyloidiasisFrom the Public Health Image Library, US Centers for Disease Control and Prevention [Citation ends].com.bmj.content.model.Caption@3e47e24c

larva currens

Rapidly moving serpiginous, pruritic urticarial rash, moving 5 to 10 cm/hour.

Can chronically recur.

Incidence varies from 10% to 70% of patients dependent on burden of infection and age.[11]

urticaria

A transient urticarial rash can occur with or without larva currens.

cutaneous larva migrans

More commonly associated with the dog or cat hookworm, Ancylostoma braziliense.

Very rarely associated with strongyloides.

apparent drug reaction rash (hyperinfection)

With hyperinfection, disseminating larvae may mimic a drug reaction, with eosinophils seen on biopsy.[21]

other skin complaints

A wide variety of presentations including disseminated purpuric rash can occur in hyperinfection.

symptoms and signs of inflammatory bowel disease

Adult larvae in the duodenum may cause a severe duodenitis evident in histology with villi atrophy and plasma cells infiltration. Colonic manifestations can clinically mimic ulcerative colitis or Crohn's disease with eosinophilic granulomatous inflammation affecting the colonic wall.

Risk factors

strong

soil exposure in or migrants from an endemic area of the world

In non-endemic regions, 99% of chronic strongyloides infections are among migrants from endemic areas, particularly refugees.[11][12][13][14][15][16] Current single-dose praziquantel and albendazole campaigns in Africa do not reduce the prevalence of strongyloides infection.[17]

The greatest risk factor is cutaneous exposure to infected soil containing strongyloides filariform larvae.

Infection is endemic in many tropical and subtropical regions worldwide and in the Appalachia region of the US and in certain Mediterranean regions, especially Catalonia, Spain.

In agricultural workers, the risk of chronic infection is increased more than 50-fold compared with that of the general population in regions where the infection is endemic.[7] Military veterans, particularly with historical deployments in southeast Asia or other tropical regions, are also at risk.[9]

corticosteroids (risk of hyperinfection)

Risk of life-threatening hyperinfection is primarily related to immunosuppression, particularly with administration of corticosteroids.[20][21]

human T-cell lymphotropic virus type-1 (HTLV-1) infection (risk of hyperinfection)

There is no association with risk of acquiring the infection; however, once infected there is risk of hyperinfection if co-infected with HTLV-1.[22][23]

weak

international travellers

International travellers are generally at low risk for strongyloides infection. The incidence of strongyloides infection in ill returning travellers attending a European travel clinic was 0.1%.[18]

However, for those with unexplained eosinophilia and negative stool specimens, serological diagnosis is recommended.[19]

impaired immunity

Haematological malignancies, tuberculosis, and malnutrition secondary to chronic strongyloides diarrhoea are all other risk factors for hyperinfection.[24]

solid-organ transplant recipient

Cases of donor-derived infection have been reported, albeit rarely, in recipients who have received organs from donors in endemic regions. Donor-derived strongyloidiasis has a high mortality rate.[10]

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