History and exam

Key diagnostic factors

common

presence of risk factors

Risk factors include bicuspid aortic valve, rheumatic fever, endocarditis, Marfan's syndrome and related connective tissue disease, and aortitis.

diastolic murmur

The absence of diastolic murmur significantly reduces the likelihood of AR.[23]

The severity of the AR correlates well with the duration of murmur, instead of the intensity of murmur. In mild AR the murmur is early diastolic, and increases in duration to holodiastolic in severe AR.

A diastolic murmur may be absent in acute AR.

Other diagnostic factors

common

dyspnoea

Caused by pulmonary oedema in acute AR, or progressive left ventricular dysfunction in chronic severe AR.

fatigue

Symptom of chronic AR due to progressive left ventricular dysfunction.

weakness

Symptom of chronic AR due to progressive left ventricular dysfunction.

orthopnoea

Symptom of chronic AR due to progressive left ventricular dysfunction.

paroxysmal nocturnal dyspnoea

Symptom of chronic AR due to progressive left ventricular dysfunction.

pallor

Sign of cardiogenic shock.

mottled extremities

Sign of cardiogenic shock.

rapid and faint peripheral pulse

Sign of cardiogenic shock.

jugular venous distension

Sign of cardiogenic shock and congestive heart failure.

basal lung crepitations

Sign of pulmonary oedema.

altered mental status

Sign of cardiogenic shock.

urine output <30 mL/hour

Sign of cardiogenic shock.

soft S1

May be soft due to early coaptation of the mitral valve leaflets from increased end-diastolic pressure.

soft or absent A2

Caused by inadequate closure of aortic valve in severe AR

collapsing (water hammer or Corrigan's) pulse

Arterial pulse shows rapid rise and a quick collapse resulting in widened pulse pressure >50 mmHg.

cyanosis

Sign of acute AR.

tachypnoea

Sign of acute AR with pulmonary oedema.

displaced, hyperdynamic apical impulse

Present on chronic AR with left ventricular enlargement.

uncommon

chest pain

Most common in chronic AR, although can be presenting symptom in acute AR. Acute severe central crushing pain may indicate myocardial ischaemia, or, if referred to the back, aortic dissection.

pink frothy sputum

Sign of pulmonary oedema.

wheeze (cardiac asthma)

Sign of pulmonary oedema.

additional heart sounds

Left ventricular dysfunction can result in S3 gallop or occasionally S4 due to left ventricular hypertrophy.


Third heart sound gallop
Third heart sound gallop

Auscultation sounds: Third heart sound gallop



Fourth heart sound gallop
Fourth heart sound gallop

Auscultation sounds: Fourth heart sound gallop


arrhythmias

May be present in acute severe AR or chronic AR.

ejection systolic flow murmur

Sometimes associated with moderate to severe AR. The murmur occurs after S1 due to the flow of increased stroke volume across a non-stenotic aortic valve. It is an early peaking, crescendo-decrescendo systolic sound, best heard at second right intercostal space, and can be differentiated from an aortic stenosis murmur by the absence of an ejection click.

Austin Flint murmur

A soft, rumbling, mid to late diastolic murmur heard best at the apex. It is produced by the abutment of an aortic regurgitant jet against the left ventricular endocardium.[20] An Austin Flint murmur is distinguished from the murmur of mitral stenosis by the absence of an opening snap and loud S1. It is a specific finding for severe AR.

systolic thrill

May be palpable over the base of the heart or suprasternal notch due to increased stroke volume.

Hill's sign

Systolic pressure over popliteal artery exceeds brachial systolic blood pressure by >60 mmHg. Peripheral haemodynamic sign associated with a bounding pulse and systolic hypertension of chronic severe AR.

bisferiens pulse

Double systolic arterial impulse.

de Musset's sign

Patient's head may bob in time with each heart beat. Peripheral haemodynamic sign associated with a bounding pulse and systolic hypertension of chronic severe AR.

Muller's sign

Pulsations of the uvula.

Traube's sign

Pistol shot sounds over the femoral artery with compression. Peripheral haemodynamic sign associated with a bounding pulse and systolic hypertension of chronic severe AR.

Quincke's sign

Subungual or lip capillary pulsations due to the large stroke volume. Peripheral haemodynamic sign associated with a bounding pulse and systolic hypertension of chronic severe AR.

Duroziez's sign

Systolic and diastolic murmurs heard over the femoral artery when compressed proximally and distally, respectively. Peripheral haemodynamic sign associated with a bounding pulse and systolic hypertension of chronic severe AR.

Mayen's sign

Diastolic drop of blood pressure >15 mmHg with arm raised.

Lighthouse sign

Blanching and flushing of forehead.

Becker's sign

Pulsations of retinal vessels.

Landolfi's sign

Alternating constriction and dilation of pupil.

Rosenbach's sign

Systolic pulsations of liver.

Gerhardt's sign

Pulsatile spleen.

Lincoln's sign

Pulsatile popliteal artery.

Sherman's sign

Dorsalis pedis pulse is unexpectedly prominent in age >75 years.

palmar click

Palpable systolic flushing of palms.

syncope

Rare presentation.

Risk factors

strong

bicuspid aortic valve

This congenital abnormality accounts for most of the cases of AR in developed countries.

Some pathological abnormalities of bicuspid aortic root occur, which lead to proximal aortic dilation and worsening of AR.[8][11]

rheumatic fever

One of the most common causes of AR in developing countries.[1]

endocarditis

Can lead to rupture of leaflets or even paravalvular leaks.

Vegetations on the valvular cusps can also cause inadequate closure of leaflets, resulting in leakage of blood.[7]

Marfan's syndrome and related connective tissue disease

80% of Marfan's patients present at an early age with a diastolic murmur.[12]

Disorders such as Marfan's syndrome often lead to progressive dilation of aortic root resulting in AR.

aortitis

Inflammation of the aorta secondary to systemic diseases such as syphilis, Behcet's, Takayasu's, reactive arthritis, and ankylosing spondylitis results in weakening of the aortic root and dilation.[14]

weak

systemic hypertension

Can lead to aortic root dilation and inadequate closure of aortic valve leaflets.[9][13]

older age

Older patients are more prone to develop AR along with aortic sclerosis.[15]

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