Traumatic brain injury (TBI) may result in a wide array of cognitive deficits, including executive functions. A proposal by Ferretti and colleagues (2011 & 2013) has suggested that executive dysfunction caused by traumatic brain injury (TBI) impact navigation and discourse abilities in similar ways, suggesting that these skills may share similar neural pathways. The present study is a novel approach to the investigation of this proposal. Two experiments were undertaken. Study A investigated overall cognitive abilities, (memory, attention, language, processing speed, and visuospatial ability), and examined group differences between adults with moderate to severe TBI and controls on both narrative and procedural discourse tasks. Study B focused on examining differences between these two groups on self-perceived wayfinding abilities as they relate to navigation/orientation, spatial anxiety, and distance estimation. In addition, the potential effect of familiarity to wayfinding in adults with TBI and controls was also examined. In Study A, planning, monitoring, and repair behaviors in navigation and discourse tasks were assessed in individuals with moderate to severe TBI and with a non-brain injured group, with three additional cognitive assessments administered to provide a baseline of cognitive abilities. Group differences were revealed in two out of the three cognitive assessments with the TBI group showing deficits in language, immediate memory, delayed memory, attention, and decision making. The discourse tasks revealed poor performance on planning, monitoring, and repair in the TBI group specifically in the story grammar structure and accuracy of productions. In Study B, no group differences were observed between self-reported navigation/orientation, spatial anxiety, and distance estimation abilities based on the findings from the Wayfinding Questionnaire. However, significant differences were revealed between the reported scores of the TBI and NBI groups in response to familiar and unfamiliar route-based questions. This work provides a foundation for further exploration of the executive functioning deficits caused by TBI and how those deficits impact other, high-level cognitive functions such as discourse and navigational abilities. [The dissertation citations contained here are published with the permission of ProQuest LLC. Further reproduction is prohibited without permission. Copies of dissertations may be obtained by Telephone (800) 1-800-521-0600. Web page: http://bibliotheek.ehb.be:2222/en-US/products/dissertations/individuals.shtml.]