Prenatal alcohol exposure (PAE) is associated with profound and lifelong disability. The umbrella term fetal alcohol spectrum disorder (FASD) describes a spectrum of impairments resulting from the deleterious effects of PAE (Chudley et al., 2005). While the core aspects of the condition remain unchanged, the Australian guidelines comprise two diagnostic categories for FASD: (1) FASD with three sentinel facial features; or (2) FASD with less than three sentinel facial features. Understanding how to support children with a diagnosis of FASD is particularly important given the condition has been linked to a range of poor outcomes, including increased contact with the justice system, substance misuse (Burd, Fast, Conry, & Williams, 2010), antisocial and delinquent behaviour, learning disabilities, externalizing, and aggressive behaviour, as well as a range of other adaptive functioning and mental health problems (Bower & Elliot, 2016; Kodituwakku, 2009; Rasmussen, Andrew, Zwaigenbaum, & Tough, 2008). There is growing evidence that a core deficit underpinning many of these adverse outcomes is impairment in executive functions (EFs; Khoury, Milligan, & Girard, 2015; Kodituwakku, 2009). EFs are higher-order mental processes which allow individuals to deploy attention strategically, hold and manipulate goal-relevant information and consciously enforce goal-directed behaviour (Baggetta & Alexander, 2016; Diamond, 2013). Given the compromised outcomes associated with EF deficits, and the frequency of EF impairment in children with FASD, this rigorous synthesis of the effectiveness of available interventions offers great value to practitioners, individuals with FASD and their families.